Infrared Sauna Structured Water Key to Preventing Heart Disease

Infrared Sauna Structured Water Key to Preventing Heart Disease

Insights from Dr. Stephen Hussey on heart disease, focusing on the roles of infrared saunas and structured water in cardiovascular health.

Infrared Saunas and Heart Health

Regular sauna use has been associated with several cardiovascular benefits:

• A study highlighted that frequent sauna bathing (4-7 times per week) is linked to a 50% lower risk of fatal heart disease, a 60% lower risk of sudden cardiac death, and a 51% lower risk of stroke.
• The Mayo Clinic notes that infrared saunas may help in treating conditions like high blood pressure and heart failure, though more extensive studies are needed.

Structured Water and Cardiovascular Health

Structured water, also known as hexagonal or magnetized water, is theorized to have a unique molecular arrangement that could offer health benefits. However, current scientific evidence does not substantiate these claims:

• Healthline reports a lack of high-quality human studies supporting the purported benefits of structured water.
• The concept suggests that structured water forms a protective barrier in arteries, potentially preventing atherosclerosis, but this remains unproven in scientific literature.

Conclusion

While infrared sauna use shows promise for cardiovascular health, the concept of structured water lacks scientific validation. Individuals should consult healthcare professionals before making health decisions based on these modalities.

For a deeper understanding of structured water and its proposed role in circulatory health, you might find this video informative:

If you enjoy hearing all about heart disease with Dr. Stephen Hussey, I recommend you check out my conversation with Dr. Malcolm Kendrick, which you can find here 👉    • The REAL Cause of Clogged Arteries | ...  

Dr. Steven Hussey is a chiropractor, functional medicine practitioner, speaker, author, and health coach. He specializes in helping people with heart disease, type 1 diabetes, and autoimmune conditions.

00:00 - Intro
01:01 - The biggest myths about heart disease
13:53 - What causes damage to our arteries?
23:35 - Stephen’s massive heart attack at 34 years old
42:38 - Doctors are handcuffed by the standard of care
48:42 - A heart attack can happen with ZERO blockage
1:03:49 - Structured water and heart disease, a fascinating connection
1:13:35 - The heart is NOT a pump
1:28:15 - The best ways to structure water in your body
1:41:22 - The REAL energy currency of the body (Not ATP)

Transcript

0:00
Life is just all about harvesting energy from the environment and using it to create order. And if you don't have enough energy in the system to maintain the order,
0:08
you get disease. Things start to break down. And so the first place that the body seems to sacrifice is
0:13
the lining of the arteries. I had a massive heart attack at 34 years old. It was a 100% blockage of the left anterior descending artery.
0:19
No signs or symptoms. No diagnosed heart disease before that. And so we should be focusing on ways to protect the lining of the artery
0:26
and how we keep blood moving and preventing clotting from happening. Instead, we've been distracted by the lipid biomolecules in the bloodstream
0:33
because of the story that "Ancel Keys" gave us of cholesterol being the cause back in the 50s and 60s.
0:38
And so if we want to protect the lining of the artery from damage, we build structured water because nothing can penetrate it.
0:45
I've really delved into heart disease and atherosclerosis and heart attacks, and what I've learned has just completely blown my mind.
0:51
I feel like I should share the information at least so people can make more educated health decisions,
0:56
because I know they're not going to get this information from Western medicine. What would you say are the most common misconceptions people have,
The biggest myths about heart disease
1:04
when it comes to the heart and heart disease? Well, there's many, and I would say the main one that people are familiar with probably is
1:12
this whole cholesterol theory of heart disease. I think that is the biggest myth around, the biggest distraction, I would say, from heart disease.
1:21
But also things like the heart is the mover of the blood or the main mover of the blood,
1:26
which I think is also a big misconception. -- - And then the actual roles of the heart... if it's not to move blood, then, then what would it be?
1:34
We've misunderstood what the role of the heart in the body is... But I think the conversation around
1:40
animal foods and heart disease is also a big myth, and they are not the cause of heart disease,
1:46
nor do they contribute in any way. ---- Yeah, I think those are the big ones.
1:52
All right, you mentioned cholesterol right off the bat. Let's jump into that and talk about...
1:57
classically what people... consider is happening there and then how you see it. Yeah.
2:03
So I guess the traditional theory or thinking, I guess the mainstream thinking is that you get high cholesterol,
2:10
or what we're really talking about is lipids, so blood lipids. So people know of "LDL" and "HDL" and things like that.
2:18
--- And those things get high in the blood and that they somehow get stuck to the lining of the artery
2:24
and they build up this plaque formation. And that has just never really been shown to be true.
2:31
There's lots of studies out there that, if you look at them, are heavily funded and
2:36
I would say influenced by industry and industry, different industries that want that theory to be true
2:43
for financial gain. - And most of the studies suggesting that suffer from that issue.
2:50
But - there's the blood is not, the lipids aren't happening in a vacuum in the blood...
2:56
So I kind of approach this logically first. There's many other things in the blood. There's water.
3:02
The blood is half water. - - There's electrolytes, there's clotting factors, there's red blood cells.
3:08
There's a glycocalyx. There's all these different things in the blood and in the artery. And so
3:14
I guess, think about heart disease as this one substance in the blood, these lipids in the blood that carry cholesterol.
3:20
That doesn't make sense. We're ignoring all the other things there. So this is something that's happening in the artery,
3:26
within the blood or near the blood, and we're ignoring everything else. And we think, oh, it's just this thing.
3:32
And it reminds me of this experiment that was done in 1886 or 1887, I can't remember which one, called the "Michelson Morley" experiment.
3:40
And in this experiment, they were trying to test and see if there was an aether, which
3:46
is basically just the empty space there. That what's consisted of the empty space around us. -- And so they were trying to see if there was a medium
3:53
through which light could travel and that it needed to travel. And in that experiment, it was.
3:58
- The results of that experiment were misinterpreted, and they said, oh, no, aether exists.
4:04
And that was, a lot of physicists at the time were like, oh, yes, this is awesome that it doesn't exist. That makes it way more easier to do my
4:11
mathematical equations. But I don't have to account for an aether. - Or a medium through which light has to travel.
4:17
And it's the same thing with this cholesterol theory. -- If we ignore everything else
4:22
there and we just say, oh, lipids, it made it very easy to say, or to develop a theory that these lipids infiltrate the lining of the
4:30
artery and cause plaque to form. - But there's major issues with that theory.
4:36
One is that when you look at what plaque is made of it's clotting tissue, the vast majority of it, I'd say 87%, based on some studies, plus or minus 8%,
4:45
depending on which person looked at it was clotting tissue, and there was very little cholesterol present.
4:52
So if it's not cholesterol, then why are we saying cholesterol is driving this disease process?
4:57
And I've had people tell me, well, cholesterol initiates the issue, and then the plaque forms after, the clotting tissue forms afterwards.
5:05
And that's all well and good, but we have plenty of literature that shows that platelets and clotting factors
5:12
initiate atherosclerosis and play a role throughout the entire process of it.
5:17
So it's this clotting tissue that forms. -- And so there's no situation where a lipid, an "LDL" molecule says, okay,
5:25
I'm going to go embed myself into lining the artery today, because that's just what I do.
5:30
And if you look at the entire environment of the arteries and you realize that there's water there,
5:35
and I'm sure we'll get into structured water and what that is and how structured water, how some of the water in the blood actually
5:43
kind of congeals itself or forms this gel in the lining of the arteries, and that nothing can penetrate that gel.
5:49
Based on the properties of this water, then if you have intact, structured water there, then nothing's going to penetrate it.
5:55
Lipids are way too big to get even close to the artery wall. Now, if there's damage, then, yes,
6:01
lots of things can access the artery wall, but it's the response to damage that causes plaque to form this clotting tissue.
6:08
So you gotta think of it like the same thing that happens if I cut my skin. If there's damage to something,
6:14
my body responds by developing clotting tissue, a scab. Because it can't heal that instantly,
6:19
it has to stop the bleeding so that the body can heal it over time. And when we get damage to the lining of an artery,
6:25
a similar thing happens. We get clotting tissue that forms and stops that artery from becoming too damaged
6:31
so that it doesn't rupture, because that would be internally bleeding, and that's a much bigger problem.
6:37
And so it develops this plaque, and the inability to heal the artery is what leads to the development of more and more plaque.
6:44
So there's a reason that we see type 2 diabetes being the number one risk factor for development of atherosclerosis,
6:51
because with type 2 diabetes, you see impaired wound healing. And so if you can't repair the wound on the inside of the lining artery,
6:58
it sticks around and we get more and more plaque development. And so we should be focusing on ways to protect the lining of the artery and how we keep blood moving and
7:07
preventing clotting from happening. That should be the main focus of heart disease. And instead,
7:12
we've been distracted by these lipid biomolecules in the bloodstream because of the story
7:17
that "Ancel Keys" gave us of cholesterol being the cause back in the 50s and 60s.
7:23
And we've been stuck on that idea ever since. I want to really get in here and differentiate where
7:30
these two stories diverge. We would all agree that there are these particles in the blood,
7:37
there is damage happening in the artery. Where is that process different between the two paradigms?
7:44
--- Well, there's some people out there that say this response to retention theory, where there's damage that happens.
7:53
But there's some people that say that no damage. I was listening to a lipidologist the other day that somebody
7:59
asked me to listen to and asked my opinion of who said that no damage has to be present
8:04
and lipids can enter the lining of the artery regardless, especially “APOB”, they say.
8:09
--- And that's just not true if you understand the fact that structured water forms in the lining of the artery,
8:16
and this structured water protects anything from getting there. So if you have healthy arteries,
8:21
there's no way any biomolecule is going to touch it, aside from very small hydrated ions of minerals.
8:27
So that's the thing is that, again, going back to the fact that they're acting like
8:32
the lipids are the only thing that matter, and they're ignoring... everything else that happens in the artery,
8:38
in the blood. And without that information, how can you have a full idea of what atherosclerosis is if you're ignoring everything else,
8:47
and that's the main issue for me, is that... they're ignoring all that stuff and they're just not looking at different information.
8:55
So that's where it really differs, is that you have to look at
9:01
this as a complex system with many multiple factors. And when you do that and you account for all those factors,
9:07
then you see this other path. Because in the 1950s, when heart disease was becoming an issue,
9:13
coronary artery disease, because in the early 1900s, it wasn't really an issue. It was very rare for have coronary artery disease...
9:21
And then in the 50s, President "Eisenhower" famously had a heart attack while he was in office...
9:26
And everybody was scared about this new disease. -- And so they came up "Ancel Keys" and his team
9:31
decided to give people an answer. And they did some poorly done research. At the time, there was epidemiology that just associated cholesterol with heart disease.
9:40
And they cherry picked the data. So they only picked the data that gave them the correlation that they wanted,
9:47
and they ignored everything else that happens in blood... And so that was a huge issue but at that time,
9:53
which many people don't know, there was also a scientist named "Meyer Texon" who
9:59
was adamant about this disease being basically a clotting issue due to interrupted or poor blood flow, which is what happens.
10:06
Like “Rudolf Virchow” back in 1856 told us that what causes clotting is poor blood flow, damage to the lining of the artery, and sticky blood,
10:14
blood that's too viscous. And so when you think about that and you realize that even during the early
10:21
research into the heart disease and this new epidemic we were having, there were people saying that, look,
10:26
I can cause atheroscosis by interfering with blood flow and creating clotting. -- But there was all this money behind this cholesterol theory
10:34
because the pharmaceutical industry had a drug that could lower cholesterol. So they were putting a lot of money behind that theory so
10:40
that they could sell drugs, the food industry, processed food industry wanted that theory to be true because then they could demonize saturated
10:48
fat and cholesterol in animal foods, and you could eat more processed foods, processed carbohydrate foods.
10:53
And so there was money behind this theory. And there's very documented evidence that money was given to support this theory.
10:59
And so it just kind of took off despite this other evidence that was there. And now we're stuck in that.
11:05
So really, the way that these two theories differ was financially a lot. A lot of it was financial push for them.
11:11
And if you look at the early studies that "Ancel Keys" and many other, there's probably five or six studies that were done
11:17
in the 60s and 70s that were testing -- saturated fat consumption and cholesterol levels versus
11:23
actual interventional trials where they replaced saturated fat in the diet with unsaturated fat.
11:29
And they found every single one of those trials found that the more unsaturated fat, the less cholesterol you ate, the more heart disease people had.
11:38
So there was never any research to back this up in the first place. So I find it very, very interesting that
11:43
these days you have to provide so much evidence against the theory to prove that it's not right when there was never evidence in the first place.
11:52
So, yeah. So you mentioned this one lipidologist that believes that just having the presence
11:59
of these substances in the blood is enough to initiate this whole cascade..When it comes to conventional thinking,
12:06
where it's assumed that there is some kind of damage there to initiate everything.
12:13
is there agreeance between the way you look at it versus conventional of what's causing that damage?
12:20
Yeah, I would say so. -- They acknowledge that there are things that can damage the lining of the artery.
12:27
The big issue I have is they're saying that no damage has to take place for lipids to be deposited in the artery,
12:34
which in my opinion, that never happens, regardless of damage anyways. It's clotting tissue that forms on the artery when there's damage.
12:43
But yes, I have heard many people on both sides say that insulin resistance is an issue.
12:49
Being insulin resistant, being diabetic or even pre diabetic or before that you can be insulin
12:55
resistant well before you're diabetic, or various toxins or stress
13:00
can all be as inflammatory things for the lining of the artery. -- And I think there is some agreeance on that,
13:06
that those types of things can be. But I would argue also that... a bigger role
13:11
is played by the things that interfere with the body's ability to heal the damage once it's happened.
13:17
That's what leads to this plaque development over time. Because the body doesn't want that there.
13:23
-- It's lost the ability to... heal it. Which is largely due to the fact that we have an epidemic of poor metabolic health
13:31
in diabetes. But even if you're not diabetic having poor metabolic health, high insulin, high leptin, but also disrupted circadian rhythm,
13:39
which is a huge issue. And there's tons of literature showing that artificial light environments
13:44
disrupting our circadian rhythm are leading to that metabolic issue. So that's what's interfering with the body's ability to
13:51
heal it once it's happened. All right, we're going to move into the way you see things with the
What causes damage to our arteries?
13:56
structured water here in a bit. But I really want to make sure, before we get into this,
14:02
we've laid the groundwork versus the way you're seeing it and conventional medicine.
14:07
And the other piece I really want to make sure we dive into is... when there is damage...
14:12
and plaque forms, how you see that happening versus conventional. Well, so they would say that,
14:18
I guess conventionally they would say that, like I said, they would say there's no damage has to be happening and that lipids,
14:25
if they're high enough or they're too high in the blood, they just go and they just find their way in between the endothelial cells.
14:32
And if enough of them do that, we develop this fatty streak is what they call it. And
14:38
to me, that fatty streak we're seeing is just damage to the lining of the artery. - And so then the body responds to that,
14:43
and more plaque or more cholesterol gets deposited in there, and it just builds up this cholesterol fatty deposit
14:49
because there's too much cholesterol in the blood. That's what they would say. And that slowly this keeps growing and growing and growing and that it gets
14:58
bigger and bigger and blocks an artery. And that's, I guess, generally their theory. And they would say that certain
15:05
lipoproteins do that more readily. There's a lot of evidence now coming out that "LDL" is not the cause of that.
15:12
So the lipidologists are saying, oh, yeah, yeah, it wasn't the "LDL." It's a specific subset of “LDL” which is “APOB”.
15:18
That's the one that's the problem. But again,
15:23
we'll talk about why that can't be when I talk about how I lay it out for myself. But, yeah, so they're all focusing on the lipids,
15:31
and they're saying that they can infiltrate the lining of the artery. And if there's too many of them, that doesn't make sense.
15:39
To me if lipids are high in the blood, first of all, when you take a lipid test,
15:45
that was what it was at one snapshot in time. So when you look at your lab test that's
15:51
what it was. It's not what it is right now. And "Dave Feldman" and many others have shown that your lipid levels change drastically
15:57
throughout the day, especially throughout the week. So that's one snapshot in time. Your lipid levels are always moving because they're based on our metabolism,
16:05
and our metabolism changes all the time based on our environment. - But they would say that -- they go in there and they can infiltrate
16:12
the lining of the artery. But if that's the case... and lipids are high in the blood, let's say
16:18
they're high everywhere in the blood. So why do we see atherosclerosis more in some places and not others?
16:24
-- Atherosclerosis -- only occurs in arteries. It doesn't occur in veins, and there's lipids in the veins.
16:30
So why don't we see atherosclerosis in veins. And people will say, because veins can't develop atherosclerosis.
16:37
Well, when you take a vein and do a bypass surgery, guess what happens to that vein? It develops atherosclerosis. So it can't be
16:44
just merely the fact that there's lipids there. There has to be something else. And I would say the lipids are largely irrelevant and they're
16:52
the wrong biomarker to be looking at. So, yeah, that's their way of thinking. So do you want me to outline my way
16:59
what I think? Yeah. Let's give a specific example of your way of thinking. Just hypothetical.
17:04
Create an initiating factor and then take me through the process of what's happening.
17:09
So I'll give a very specific example of initiating factor, because it happened to me.
17:15
And this is not something that's in my book. So this is a bonus. One thing I wish I could make it longer. So
17:22
I had a heart cath done, and after the heart cath, so they went in through the artery in my leg,
17:28
in my groin, the femoral artery, to do the heart cath. And... my leg was fine... the morning of the heart cath, before the heart cath,
17:36
because I did a sprint workout, and everything was fine. My leg felt fine. -- And then about a month after that heart cath,
17:43
I was noticing that I was getting pain in my lower leg, so below the knee, and didn't know what was going on for a long time.
17:50
I was like, what is this? Is it "DVT?" Is it a compartment syndrome? What's going on here? And eventually
17:56
found out that because of the heart cath and the device they used to seal the artery after the heart cath,
18:03
I developed plaque in my leg, in the upper part of or the lower part of my thigh, in the femoral artery,
18:09
which is a very common place for it to happen. And so the only thing that happened there that was different
18:15
was the change in blood flow dynamics that happened going into my leg. And so as this device was sitting there,
18:21
and part of it's on the inside of the artery. So when the blood's flowing past it, it kind of disturbs it and messes with it and makes it eddy and that kind of stuff.
18:30
And so it was pooling up in that area, and that pooling up developed clotting tissue.
18:35
It's this lack or interrupted lack of blood flow or interrupted blood flow that caused it. And it developed it right there downstream from where that device was.
18:43
And it happened very fast because I've had many people tell me that these studies on people with high "LDL" after five or six years and they don't have plaque.
18:51
Yeah, well, it takes a long time to develop. Well, guess what? This was like less than a month when it developed for me, this interruption of blood flow,
18:59
this clotting tissue formed. And so, yeah, I couldn't even walk to the airport fast without pain in my leg.
19:05
And so that's one thing that can contribute to plaque formations, is interrupted blood flow. And like I said,
19:11
“Rudolf Virchow” told us that way back in 1856, he said this is what causes clotting in an artery.
19:16
He said damage to the lining of the artery in response to that interrupted blood flow.
19:21
And when blood gets too sticky or too viscous, when our blood is not thin, so to speak.
19:27
So that's one thing, and that's a major thing that mattered to me personally. So that's one example of what can develop clotting tissue.
19:35
But it could also be a acute toxin exposure that overwhelmingly damaged the artery...
19:40
So one thing in particular could be endotoxemia, which is when you get gram negative bacteria from the digestive tract.
19:48
Somewhere from the digestive tract, whether it's the small intestine or the mouth, due to poor dental care, things like that, you can get this leakage of
19:56
endotoxic bacteria into the bloodstream. And there's a heavy, heavy association between having that in the bloodstream and your body
20:02
attacks that bacteria and that releases endotoxins. That's what your body's supposed to do. It's supposed to attack that bacteria,
20:09
but it releases endotoxins which are incredibly inflammatory and they've been shown to damage the lining of the artery.
20:15
We're getting this acute damage due to this acute toxin exposure. So that's another thing that creates that damage.
20:20
But you can get prolonged damage too, for many different lifestyle choices.
20:26
And then, -- the third thing that "Virchow" says is when the blood is too viscous, we know that when things aren't, they're kind of sludgy.
20:34
They're not moving through as well, it's more likely to have this clotting response because that's what happens.
20:40
So a clotting response is a normal, healthy thing if it happens... physiologically, but pathologically, if it's happening too much,
20:48
it becomes a problem, especially in the lining of the arteries. And this is the part I really wanted to zone in on,
20:55
the clotting response, where in the way you see things, this is what happens when there's Injury in the vessel,
21:03
which is totally different than conventional thinking. Exactly. Yeah. So
21:08
a clotting response is something that's supposed to happen. Again, if you cut your skin and you don't want to bleed out,
21:14
so your body initiates a clotting response and stops that bleeding from happening and forms a scab.
21:20
And if that... damage to the lining of the artery happens so intensely in an acute situation, or
21:26
it just kind of wears away over time from chronic inflammation. And then
21:32
the damage happens on the lining of the artery. And if that damage continues to happen, the body doesn't do anything about that.
21:39
The artery is going to rupture. - It's going to become so weak and rupture, and then you're internally bleeding.
21:45
So the body initiates a repair response, which is the formation of clotting tissue. So you have all these clotting factors in your blood.
21:52
I remember learning about all of them in school. They're Factor 8 and all these different factors, and fibrinogen and prothrombin and all these different things.
22:00
And there's this clotting cascade, they say, and you initiate that physiologically when it's supposed to happen, then that's a normal thing that we need,
22:07
because there's normal wear and tear that needs to happen, or that has to happen and we body has to repair it.-
22:13
But when it happens excessively or pathologically... because we have damage to the lining of the artery and
22:19
our blood's just not moving well and it's too viscous, then it's just more likely that we develop this scab,
22:25
basically this fibrotic clotting tissue on the lining of the artery. And then, like I said, if you can't heal that scab,
22:32
you can't heal that damage. And there's still inflammation. It's just likely to build up and build up and build up.
22:38
Or... people have heard of plaque ruptures, and the plaque can rupture. And there's lots of evidence that plaque ruptures very rarely,
22:46
if ever cause a heart attack. But they do lead to more development... of that plaque building up over time.
22:52
But it's all a clotting response, which... that should tell us, though the answer to preventing atherosclerosis is preventing a clotting response,
23:01
not micromanaging your lipids, because we've seen what that approach has done. Statin drugs are the number one prescribed drug in the world.
23:10
They're one or two, depending on the year, from opiate drugs. So they're very
23:16
commonly taken but heart disease is still the number one killer. So if we were having really an effect, then
23:23
it would be more effective than that. We really should be focusing on clotting.
23:28
And that's what the literature shows - very robustly, is that we should be looking at clotting factors.
Stephen’s massive heart attack at 34 years old
23:35
Let's come back to your example there. With the leg, you have this clot. When did you realize what was going on down there?
23:42
Obviously you're feeling the pain you mentioned when you're walking. But when did you get definitive data on what was happening?
23:50
And then what happened from there? That was about six months. Well, maybe five months after
23:56
I initially felt symptoms, I finally went in and got a doppler ultrasound that confirmed that
24:02
there was a 75 to 99% blockage of that artery in my leg. So pretty severe. And so just so people know, I'm also type 1 diabetic.
24:10
And so this is called peripheral artery disease when you get plaque development in your leg. And
24:16
to me it was due to this device. And so the studies on peripheral artery disease show that type 1 diabetics
24:23
usually have worse outcomes than even type 2 diabetics for whatever reason.
24:28
And so it was July of 2021 when I first got that
24:33
imaging done. And so at the time. the vascular surgeon I went to see after that was
24:40
well, we could stent it or we could bypass it or whatever, but he's like, it's only interfering with things you like to do.
24:47
It's not interfering with your day to day life. So let's leave it alone, because usually when we mess with them,
24:54
they get worse. And I was okay, cool. And so I said, I'm going to do my own thing
24:59
based on information that I knew at the time. I didn't know everything that I know now by far,
25:06
but I knew some things. And so I did some different things.
25:11
My diet stayed the same, which means my "LDL" level stayed the same. My stress stayed about the same, my
25:18
exercise stayed about the same, although I wasn't as intense because I couldn't
25:23
do that without pain in my leg. I could ride a bike, though, without pain in my leg, and so...
25:29
all that stayed the same. What I changed was I increased my infrared sauna use.
25:34
I bought a sauna and did that. Got more sunlight exposure, did more grounding and set my circadian rhythm.
25:41
Those are the things that I changed. I'd never done those things intentionally before.
25:48
And in a year... I went back and got another ultrasound and it was only 50% blocked.
25:53
And the technician came back in and said, the doctor wants me to look at it again.
25:58
We got a weird result. So we did the test twice, and it showed it was only 50% blocked.
26:04
And at that time, the vascular surgeon said, well, we can't say this is better because we don't see these things get better.
26:11
Those were his exact words to me. And I was like, well, the testing just showed that it's better, but I didn't really say anything.
26:19
I was just okay. So I went back and I kept doing the stuff. And then a year after that
26:25
I went back again it was completely normal. And so then he said, we'll come back in a year,
26:30
we'll look again. And I said, okay. So I came back in a year, and that was this past the last May we had,
26:36
and it was still normal. So my leg is normal now. I can run, I can hike, I can snowboard,
26:42
and I have no pain in my leg, which is unheard of within Western medicine that they say that that's impossible.
26:48
I should not have been able to reverse that plaque and I did. And I did so
26:53
by using these modalities. And I'm sure we'll get into explaining why those modalities were helpful as
26:59
far as vascular health and allowing my body to reverse that. But
27:05
just so people get a sense of how impossible Western medicine thinks that is,
27:10
I was on "X" the other day and I posted about this stuff and there was this other influencer type doctor
27:17
that was saying to me... just something. And I said, well, hey I actually reverse plaque in my body.
27:24
And -- they were like, there's no way you did that. That's impossible. And I was just
27:30
okay, well, and I posted -- literally my chart notes, my study results
27:35
there. And he said, - -- you shouldn't read your own results. You should get a vascular surgeon reading. I was like, well,
27:41
who do you think ordered these tests for me as a vascular surgeon? This is who interpreted this. And he was
27:47
there's no way you did that. That's impossible. And I was like, here it is. So
27:53
I don't know. I just stuck in that paradigm that that's not possible, but what if it is?
27:58
As at least one example that it is. And so I think that I know the mechanisms by which I did that,
28:06
or at least I have theories about how I did that because it's super important to figure that out.
28:12
- Before we get to that, I want to fully set the scene here with your story, we've talked about the fact you're type 1 diabetic.
28:20
You had this blockage in your leg, which you've completely reversed. We got to go back even a little bit further than when this blockage occurred,
28:28
January 5, 2021, you get done an exercise routine in the morning,
28:34
and you have a massive heart attack. So take us back to that morning...
28:40
and what happened. Yeah, I've been interested in health most of my adult life,
28:47
and I could always really control diet and exercise, and that's what I did.
28:52
I was always really good about controlling that stuff. But, yeah, I had a massive heart attack on
28:59
at 34 years old. Like you said, a widowmaker heart attack. So it was a 100% blockage of the left anterior descending artery.
29:06
No signs or symptoms, no diagnosed heart disease before that. -- And I actually had a "CAC" score done, which is a
29:13
measure of how much calcified plaque there is in the arteries of your heart. I had that done six months prior to this, and it was zero,
29:20
which suggests I'm at very low risk for heart attack. It doesn't measure soft plaque, but it measures hard plaque,
29:27
and a zero score is pretty good. -- So, anyways, this happened, and the reasons that I think it happened, because when I was in the hospital,
29:35
they were like, oh, you're cholesterol, it's cholesterol, it's cholesterol. Every person I saw,
29:41
I probably had three or four different doctors or attendings or residents while I was in the hospital.
29:47
And I would ask all of them, because at this point, I was hey, tell me what you think, because
29:53
this is shocking to me. and it's what really pushed me even further down this path of
29:58
figuring out what's going on. But, yeah, their answer was very unsatisfying to me. And I knew,
30:04
I was aware of a lot of the research on cholesterol and how ineffective statins are and how the theory was not
30:10
well founded by science. So I questioned that, and I was very disappointed with their
30:16
answers. And it just really showed a lack of critical thinking or lack of
30:22
openness to different discussion. And so... my best guess, which no one will ever be able to tell me exactly what happened,
30:29
but when they looked in my arteries and when they went in to bust the clot and place the stent,
30:34
they found no atheroscosis anywhere except for the giant clot in the "LAD." So my arteries were pretty healthy.
30:41
And so what happened was an acute clot formed, spontaneous acute clot that was large enough
30:46
to block the whole artery. So you get chronically formed clotting tissue from damage to the lining of an artery
30:52
that can form plaque over time. Or in my case, you could put yourself in a situation where it's acute enough that
30:57
it blocks the whole artery... And for me, what I think did it was at the time, I convinced myself that
31:02
it didn't make sense that our ancestors drank water all day long, so you didn't need to drink that much water.
31:08
-- And so I was drinking probably about a cup of bone broth a day, which was unwise. I think I was pretty dehydrated.
31:14
And I know I was dehydrated because I was dealing with a little bit of constipation at the time, and...
31:20
I hadn't quite convinced myself to do anything about it yet. And then... I went through
31:26
a period of higher stress. "COVID" was happening, and there were things I didn't like about "COVID" that was stressing me out.
31:33
But then with all that happening. Then a day and a half before the heart attack,
31:39
I got very stressful news about a family member of mine. And it was the inability for anybody
31:45
in my family or me to do anything for this person, get to this person, that was stressful.
31:50
So Sunday night, I heard that news, didn't really sleep Sunday night. Monday was kind of this blur where I was trying to figure out if
31:57
I could get to this person. Monday night, didn't sleep very well at all. Woke up Tuesday and said, hey, gotta get my mind off of this.
32:05
And I did a very intense workout, like you mentioned, first thing in the morning. Well, not first thing, probably mid morning,
32:12
where I would sprint up a hill, drop down and do pushups to failure, do lunges to failure. My leg was fine in that point.
32:18
That was the workout that I did where my leg was fine. And then 20, 25 minutes later, after the workout,
32:24
when my body had calmed down, boom, I started feeling this pain right here in my chest. And at first it was somewhat mild.
32:30
I thought my "pec" was tightening up. I was like, oh, that workout must have been intense. But it intensified.
32:36
I started to get really flushed and hot and my body just got super warm. And I was like, oh, gosh. I went outside
32:43
because it was January. It's cold outside, and it just wasn't going away.
32:49
So I smartly called the ambulance. And, yeah, we did the intervention.
32:54
And that's what Western medicine is amazing at. In that situation, they saved my life. And...
32:59
Western medicine has saved my life three times now because of those types of situations, emergencies.
33:06
And yeah, very grateful that they saved my life. But then after the fact, what they told me to do was very, very disappointing
33:15
because I said okay, guys, what do you got for me? And it was just like a broken record.
33:21
Cholesterol. And every time I would ask him about the medications they wanted me on and why they wanted me on them, and
33:28
they were just very not open to conversation about it. -- And there was a cookie cutter recipe
33:34
of this person's had a heart attack. This is what they should be on. And so as an example,
33:39
they wanted They said I'd be on five medications the rest of my life. Two of those were blood pressure medications,
33:46
a beta blocker and an "ACE" inhibitor... And I decided to take one of those for one day. I took one dose of it,
33:52
and I remember waking up in the middle of the night. The nurse came in and checked my blood pressure and it was
33:58
90 over 50, which is incredibly low... And so I was oh, gosh, that's really low. And so then
34:04
I was up and I realized that it was hot in the room. So I went to turn the thermostat down, and when I stood up out of the bed,
34:11
I almost passed out. I kind of went black and stumbled and fell into the wall a little bit.
34:16
My blood pressure was really low. And that was only one dose of one medication they wanted me on,
34:21
too... So it was basically, again, this clear cookie cutter recipe. This is what you get. There was no critical thinking of, hey,
34:28
this person has had normal blood pressure their entire life. Maybe it would be dangerous to give them two blood pressure medications.
34:37
And I get what their reasoning was. They want, I'm on those to prevent heart failure, which
34:42
in my opinion, there's much better ways to prevent heart failure after a heart attack, which I did.
34:48
But, yeah, so just lack of critical thinking. They also told me to eat a processed food diet.
34:54
You should have seen when the nurse came in and told me what diet was best for my heart. Things like
35:00
animal crackers and "Fig Newtons" and... foods that were full of seed oils and things.
35:05
Those were the heart healthy foods. And so I was just well, this is a processed food diet. This is not,
35:12
I know what the American Dietetic Association is funded by big food. I get that. I understand all that, but this is not what I'm going to eat.
35:21
And so I quickly got reinvigorated having this experience in the hospital, knowing that this was not the direction I was going to go.
35:29
And I took a blood thinner for six months because of the stent in my heart. But after that, I'm on no medications.
35:36
And I'm not saying that everybody should do that. I'm just saying that's what I chose to do
35:41
based on my situation... And in two months, aside from having this pain in my leg,
35:47
I guess maybe it was three months, aside from having this pain in my leg, I went back and got my follow up echocardiogram.
35:56
And I'd been using an infrared sauna at that point. Not as frequently as I did after I got the leg diagnosis,
36:03
but I was using sauna and my heart had totally recovered.
36:08
As far as the conduction signal through my heart tissue and my ejection fraction, which is how much blood
36:17
leaves the left ventricle, every contraction, it was at 30 to 35%, I think,
36:23
which normal is 50 to 70%. And at three month echocardiogram, it was 55%.
36:29
So it was in the normal range. And so I went to the cardiologist one time after this follow up and...
36:35
they said, oh, you must -- have decided to take your medications. And I was
36:40
actually, no, I didn't. And I'm just an N of one example. But
36:45
he was oh, well, you need to take those. And I was okay. So it was just like this.
36:50
That was what they have for me is you need to be on this protocol, taking medications.
36:56
And since that time, I have even more than I had before because I'm predisposed to heart disease.
37:02
As a type 1 diabetic, I've really delved into heart disease and atherosclerosis and heart attacks.
37:08
And what I've learned has just completely blown my mind. And what I've been able to reverse and heal from
37:15
without following their recommendations is, they say, unheard of and impossible.
37:20
So I feel like I should share the information at least so people can make more educated health decisions because I know they're not going
37:29
to get this information... from Western medicine. Given what you went through there in the hospital,
37:35
you clearly mentioned that they saved your life, but afterwards, with the drugs, with the food,
37:42
how would you recommend somebody going through a similar health crisis and being in that turmoil where decisions need to be made quickly?
37:51
Drugs are being offered different foods, being pushed your way. It sounds like given
37:58
what you just shared, you navigated that pretty well and you have a medical background as a chiropractor,
38:04
so you have that going for you. And you understand science, and you were already living a healthy lifestyle...
38:10
But for somebody in that boat or just a layperson given that experience,
38:17
how would you recommend they navigate that world? And obviously with the caveat, everybody's
38:23
got different situations, needs to make their own decisions. But...
38:28
what would you say do differently and how would you guide somebody? Well.
38:34
I don't know that I would do anything differently. -- Yeah, looking back,
38:40
I don't think I would do anything differently. I was very inquisitive of them and just learned very quickly that they were
38:46
going to shut down the conversation. The last attending that came in the day I got discharged said
38:51
I'd never met this guy before yet and he said, well, I'm not going to ask you about medications because I've seen that
38:58
you don't want to take those, so I'm not even going to bring it up. And I was just well, maybe I'd be interested to know what you have to say.
39:04
I haven't met you before. Just get another person's opinion. But it was just shut down a conversation - if I wasn't going to do the recommended, then it was nothing.
39:12
And so I guess one piece of advice is that people who are more in the know or maybe frustrated with some of the things that Western medicine does,
39:21
most important thing is don't get angry. That's not going to help your situation.
39:26
And so. Yeah, but then also I would say that in that situation,
39:32
I was by myself. I had no support because it was during "COVID" and nobody was allowed to visit me.
39:38
So I was just on my phone researching like crazy. That's all I could do sitting there in the hospital.
39:44
So. But I would say that. - In that situation you're scared.
39:50
This thing happened to you. Especially me I was just how could this have happened?
39:55
I've been very health conscious my whole life, and I've And I've learned that diet and exercise is not going to prevent a heart attack.
40:03
It's not going to prevent heart disease, and it doesn't in a lot of people. That's because it's a way more complex disease and has much more
40:10
to do with environmental changes. But in that situation, you really have to try and not let the fear overcome you,
40:16
because fear shuts down decision making when your brain is in that fear state, you don't think very well.
40:23
You want to be told what to do to get to a more safe situation. And --- it becomes a game in your head of really
40:31
controlling that fear so that you can think critically --- and so and think that this is just one person's opinion,
40:38
this person in front of you. This is just one avenue of an approach you could take...
40:43
and it may be the approach you decide to take. And that's totally fine because ultimately
40:49
what you do in that situation is up to you. It's not up to your doctor, it's not up to your spouse or any family members.
40:56
It's up to you. It's your health and it's your body and you have to decide what you want to do.
41:02
if I was advising someone, even if they felt like they needed to take drugs or do some things that Western medicine says,
41:09
then that's fine. I would say do whatever you want to do, but also do these other things that are actually going to
41:16
create better health in you. And those are the things that I've kind of
41:22
fleshed out and discovered over the last four or five years as far as what's going to keep that healthy. And then
41:29
I would also say that as soon as you're able get out of that hospital because
41:35
it is the best place to be in an emergency and probably the worst place to be for healing.
41:41
As soon as you're stable and it's safe to leave, get out. Because when you think about
41:47
the "EMFs" in there, you think about the food they serve you, which is terrible.
41:52
You think about being indoors, away from sunlight and under artificial light.
41:59
How disrupted your sleep is. They come in three times a night and take your vitals and wake you up.
42:05
It's just like if they understood what healing actually is, they would not do that.
42:11
There's all these beeping lights and noises on in the room all night long. You're hooked up,
42:17
all this stuff like you can't sleep at all. And it's just an incredibly
42:22
I guess, an environment that's incredibly unconducive to healing. And so as soon as you can get out
42:29
and start putting yourself back in an environment that will allow you to heal.
42:37
You mentioned all the crap that they offered you food wise, including crackers. Another layer on that making that even more crazy is the
Doctors are handcuffed by the standard of care
42:45
fact you are type 1 diabetic and you're somebody that's injecting insulin and needing to manage
42:51
your blood sugar levels more than the average person, arguably. Yeah.
42:57
And that was another huge battle that was the most stressful part of being in the hospital. So, yeah, I'd forgotten that, but it's in the book,
43:04
so I'm sure you just read it. Yeah, it was incredibly frustrating because I'm type one diabetics.
43:10
I have been since I was nine years old. I didn't control my blood sugars very well as a teenager,
43:15
but ever since then, somewhere in college, I got a hold of things. And -- my "A1C" at the time in the hospital was 6.5,
43:22
which is good for a type 1 diabetic. It's been lower than that before. And so that means I have very good control.
43:29
Yet in the hospital, they refused to let me administer my own medications as far as insulin.
43:34
And they would do it on this sliding scale where they would only give me insulin at meal times,
43:39
and they would only do it based off of what my blood sugar was and the food that I was eating. And
43:45
if that didn't work, which it was not working because my blood sugar was 200, 300 the entire time I was in the hospital.
43:52
And I was guys, I need more insulin. If what you gave me at the last meal did not work,
43:57
I need to be more aggressive. And they were well, we can't do that. And I was well, let me do it. Give it to me.
44:03
And they wouldn't give it to me. It was just the most frustrating thing because here I am with proof that I can manage blood sugars on my
44:10
own and they're not able to. And it was, I'm supposed to be in this healing state and my blood sugars are 300.
44:17
And yeah, eventually, about the, I think the, a day and a half. I was only in the hospital for three days, but a day and a half in
44:24
my boss brought a backpack to the front desk and they brought it up to me, and there was clothes and stuff in it, but -- he also put my insulin in there
44:33
so I could manage it myself finally. But that was incredibly frustrating and it just goes to show that they
44:39
really don't know what they're doing for the most part, they don't know how to optimize things.
44:45
They have a protocol they have to follow. And that may work in some instances, but it doesn't work in every instance.
44:51
And there's no critical thinking. They can't because they're afraid they're going to get sued. If I'm administering my own insulin and something happens to me,
44:59
they're afraid I'm going to sue them, which I wouldn't I don't care. So there'd be no negligence there.
45:06
But it's that fear in them that shuts down their decision making and the protocols that they have in there,
45:11
that shuts down their decision making or ability to do anything. Yeah. It's a big issue.
45:16
I think it's important you brought up that last piece about negligence and being held accountable to a certain standard,
45:24
because in all fairness to them, the problem's even higher. It's the system that they're working under that...
45:31
handcuffs them basically into not being critical thinkers and just following protocols.
45:38
Yeah. And even when people go to a cardiologist or any doctor, really,
45:43
you have to understand... that they are somewhat bound to recommend what's the standard of care
45:49
and if they don't recommend that or they don't chart that they recommended that,
45:55
then technically they're liable. But what are they liable for? Well, they've been
46:00
told and the hospital systems have people on staff that tell you if you don't follow standard of care and you
46:08
demonstrate negligence or whatever, that you're liable and your career is on the line.
46:13
Because if someone sues the hospital or sues you, you could lose your license, all this kind of stuff.
46:20
And that's, to me, fear tactics. But they're operating out of fear again, which shuts down their decision making or their ability to critically think,
46:30
what should I do for this patient? Well, I've been recommending this drug for 20 years, and no one's ever really gotten better.
46:37
Maybe I should do something different. You can't think that way because that's the standard of care.
46:43
You have to do that, or you have to at least document that you said that. And if you don't, you're programmed to think that
46:50
something bad's gonna happen to your patient and they're gonna come back and sue you.
46:55
And then the hospital gets mad at you, and then maybe you lose your job, maybe you lose your license. I don't know.
47:02
But it's an unfortunate situation. And I kind of feel like there's a lot of bad doctors out there, but there's a lot of bad of any profession
47:09
out there. -- But I do think that ultimately the system works by the insurance companies,
47:15
the pharmaceutical companies, and the hospital systems really feeding each other a bunch of money
47:20
and creating a huge profit in feeding that. And then the doctors are stuck in that system also benefiting from that system
47:27
because they get paid very well, too. So when you pay them that well and they are allowed to provide
47:32
for their families very well, then the system's supporting them and they kind of like
47:37
making this much money. And so it's just kind of, they get stuck in that system...
47:43
with these standard of cares that make it less likely to feel change and everything. - And then the consumers, the patients are here on the outside,
47:51
and nothing's really ever. There's no benchmark that's saying we get paid well, we do better when the patients do better.
47:58
It's all about the profit of the system. And even the doctors and people in the system are incentivized by how much
48:05
profit the medical system makes. They get kickbacks from that, they get paid more bonuses, whatever.
48:10
And so the whole system is set up that way, and the patients are left over here. And so getting back to that one question you asked me is
48:18
what do you do in that situation? Is you just be educated on how that system works. Because if you're not,
48:23
you may incorrectly assume that the people in there are really just giving you the best advice we have available,
48:29
when in reality we don't. We have the advice that supports the system financially very, very well.
48:34
And sometimes it's the right advice most of the time it's very wrong advice as far as what's best
48:40
for a person to be healthy. All right, I want to come back to the morning of the heart attack.
A heart attack can happen with ZERO blockage
48:46
You mention at that time, no calcified plaque, you were under stress,
48:51
you were potentially dehydrated. Talk more about the physiology --- of this spontaneous clot
49:00
because this is different than anything I've heard of before, where you're a healthy guy,
49:06
no calcified plaque, and then all of a sudden you get this blockage out of quote, unquote, nowhere.
49:13
What do you think happened there specifically? Yeah. So if you look at...
49:18
the literature and you ask most cardiologists, interventional cardiologists,
49:23
open heart surgeons, anybody, they'll tell you that the vast majority of the time.
49:31
- I've had one surgeon tell me that 100% of the time - heart attacks and blockages and things
49:37
that, whether it's a slow, gradual blockage or stenosis or an acute blockage, they happen in places where there's bifurcations or
49:44
there's a turn in the artery. These places that create turbulent blood flow or interrupted blood flow.
49:50
And so remember I mentioned that guy “Meyer Texon” back in the 60s, who was figuring out basically that poor stagnant blood flow
49:58
was causing clotting to happen. So what he was saying is that if we have poor blood flow through an area,
50:04
the body tries to build up the endothelial cells lining, trying to make a narrower space, and the artery can kind of constrict because -- if you put
50:12
your thumb over a fire hose, it comes out more quickly. It increases the flow. And so that's what the arteries are trying to do.
50:19
But it creates that proliferation that narrows the space, and that
50:25
A, creates this proliferation that can become clotting tissue. But B, it creates a narrower space.
50:31
So if there is thick blood or there's a turn in the artery, it's just more likely that this blood starts to get stagnant
50:39
and that stagnant, when blood is stagnant. So if you get an injury to the lining of an artery
50:45
let's say an acute injury. If I cut my skin, that interferes with blood flow dynamics
50:52
because now we got breaks in these tubes or we got rough areas. - And that interference of blood flow dynamics is what triggers
51:00
that clotting response. And this clotting cascade has a clotting response. So... to me,
51:05
exercise is an inflammatory thing and generally it's a hormetic stress, this kind of acute inflammation that results in a net positive because
51:13
you train your body to be better. However, in the state of dehydration, where there's
51:20
more viscous blood, there's not water in the blood or end in acute stress, which, if you look at acute stress,
51:27
effects on clotting factors nine different clotting factors skyrocket under acute stress...
51:32
Which makes sense because if you're perceiving stress, your body thinks there may be an injury,
51:38
so it's getting ready to clot if there's an injury and stop you from bleeding.
51:43
So my clotting factors are probably elevated. - And then I did this inflammatory thing that's this generally
51:50
would be a good thing, but maybe not in that state. And then as soon as my blood flow slowed from the exercise,
51:57
a clot formed. And it formed in the artery under the most pressure of any artery in the entire body,
52:03
which is why we get plaque in the coronary arteries more commonly than any other arteries.
52:08
Because when there is things that damage the lining of the artery the stress I was going through,
52:15
then it gets pushed up against the lining of those arteries more. And there's also a lot of bifurcations and turns.
52:22
Not bifurcations, but there's turns that are very convoluted, those arteries. And so that's exactly where it happened for me.
52:29
There's just one spot near the very top of the "LAD" that has this turn in a lot of people, and that's exactly where it happened to me.
52:36
So we had this acute inflammation, and there may have been inflammation to other places in the arteries too,
52:41
but it just didn't result in a clot trigger being triggered. And so that's what happened
52:47
that's the physiology of the blood initiates a clotting response. And in my case, it was large enough
52:53
that it blocked the entire artery, but it could have just been enough that it created this little plaque formation on it.
52:59
- And if maybe I was more hydrated or maybe if I was at the time, if I was under infrared light or if I was grounded or something,
53:07
maybe it wouldn't have happened. I have no way of knowing that, and maybe it still would have happened.
53:13
But those are things that promote blood flow and adequate motion to the blood.
53:20
That's to me what happened. But again, no one will ever be able to tell me what really happened.
53:26
Even then, just to kind of challenge people because this is the way my brain thinks. It's like... they go in there, do the heart cath.
53:33
They go in, they're visualizing the arteries, they see this blockage, and they see that there's interrupted blood flow and they see it on the angiogram
53:41
- and they say, well, how do they know that blockage wasn't there prior to the heart attack?
53:47
They don't, because this is the first time anybody's seen it. And there are mechanisms of heart attacks that happen with no blockage whatsoever.
53:55
So how do I know it wasn't that type of heart attack? I don't and no one does and no one ever will. I do know, though,
54:01
that it likely wasn't that type of heart attack, because when they intervened and they busted the clot, my symptoms went away.
54:07
But I also know they gave me morphine. So maybe I just didn't feel anything because of the morphine and my nervous system calmed down and I stopped having that heart attack response.
54:16
So these are all things I talk about in the book, these different mechanisms that we could get heart tissue death.
54:22
But I don't know that it wasn't those things. But it doesn't matter to me because
54:28
I know enough now to know that whether it was a blockage or a type of heart attack without a blockage, I know how I'm going to get up and live my life.
54:35
- I know what I'm going to do every day to put myself in a situation where...
54:40
I don't have that again. And up until now, it has healed arteries in my body.
54:46
This is what I've been doing. So I'm going to keep doing that... And -- that device in my leg,
54:53
it's still there. So if I stop doing these things, maybe that plaque comes back,
54:59
I don't know. But I'm not going to try that out. -- Come back to that type of heart attack that somebody can have
55:06
without any blockage whatsoever. Because before coming across your work, I hadn't heard of that.
55:12
Yeah, in medicine, they document these, as they call them “MINOCAs."
55:18
So “MINOCA” stands for myocardial infarction with non obstructive coronary artery,
55:23
which means there's no blockage, but we have heart tissue death, so how could that be?.. And so -
55:30
there's this work of a guy named "Giorgio Baraldi," who, his research is largely ignored by the field of cardiology.
55:38
But he basically shows that a blockage of an artery, a gradual stenosis over time,
55:43
or a plaque rupture are not really the causes of heart attacks. And
55:49
we have evidence now. I have not been able to find a study that shows that treating that lesion, that gradual stenosis
55:55
with an elective stent, not in -- a heart attack situation, but an elective stent where there's no heart attack happening and they go and
56:03
they place a stent or a bypass surgery is useful as far as preventing a heart attack in the future.
56:09
Those procedures just don't work. Whether you have them or not doesn't matter. You're just as likely to have a heart attack,
56:16
according to the studies. And so "Baraldi's" work showed us that those stenosis, they build collaterals.
56:21
Whenever you get to about 60 to 70% narrowed of an artery, the body always builds collaterals. I've had cardiologists tell me that only some people do.
56:29
But "Baraldi's" work showed that 100% of the time, and he autopsied thousands of hearts during his career
56:35
that they form the collateral arteries. So that's why the stents and bypasses don't really make a difference because
56:41
the body's already figured it out. But then he developed a theory, and there's others that have
56:46
come up with this theory too. And there's lots of evidence to suggest that these steps do happen,
56:52
of how we get heart tissue death when there's no blockage of blood flow. And it has to do with
56:57
the amount of stress signaling we get to our heart. So the nervous system, we think of it,
57:02
people think of it as an autonomic nervous system that has two parts. It has a -- stress and a non stress signal.
57:08
And in reality, we're never, people think we're in one or the other, we're in sympathetic or parasympathetic,
57:15
but we're really never in one or the other. The body's always sending signals of both and they're balancing each other out.
57:21
So if I have a stress response and I get a surge of adrenaline and stress to my heart, then there's always supposed to be...
57:29
a lesser balancing signal of non stress to balance things out. So people could think of it like this.
57:35
If I'm walking my dog on a leash and my dog has a stress response because it sees my neighbor and
57:42
it wants to go after my neighbor, the dog has the response and it goes. But then I have a leash, and that leash is the non stress response,
57:50
holding things in place, stopping something catastrophic from happening... If the non stress response is not there, there's no leash.
57:58
My dog attacks the neighbor, I get sued. - That's the catastrophic event. But the parasympathetic, the non stress signaling is holding that,
58:06
keeping that from happening. So that's what's supposed to happen in the heart. However, we can get into a situation
58:12
where... that doesn't happen. We get a surge of stress response without the lesser surge and balancing non stress response.
58:19
And that non stress response signaling, what's critical for it to get into the cells, the heart cells, is
58:25
nitric oxide. So if you have poor endothelial function, poor artery function, low nitric oxide,
58:31
higher blood pressure is a sign of that, or poor metabolic health could be a sign of that.
58:37
Then that non stress signal doesn't get into the heart as well as the stress signal...
58:42
So you get this surge of stress signal into the heart cells. - And the heart typically prefers to burn fatty acids or ketones.
58:50
And there's tons of literature on that showing that even in the presence of glucose, which is
58:56
peculiar based on the rest of the body, the heart will preferentially burn fatty acids and ketones
59:03
because they're more efficient fuel sources. And the heart is always contracting, so it needs more efficiency.
59:11
But however we get that surge in stress signal, it causes this increase of burning of glucose -- in the heart cells.
59:18
So this is similar to if you went for a run and your muscles start burning glycogen in your muscles.
59:24
When you burn glucose glycogen, you get a lactic acid buildup. And a hydrogen ion buildup from burning that
59:31
glucose. And that can create a muscle burn in your leg. We feel the muscle burn, and then we stop.
59:37
We just stop running, and it goes away. The body gets rid of that lactic and hydrogen ion buildup.
59:44
However, in the heart, if that surge of adrenaline happens, it starts burning more glucose and we start to feel a little discomfort,
59:51
which I think is what angina is. It's not always a lack of blood flow to an area.
59:58
It's the heart burning more glucose than it wants to. We build up that lactic acid. And that's a problem because the heart just can't stop contracting.
1:00:06
-- We can just stop running and get rid of that. So what happens is that buildup of lactic acid and hydrogen ions creates
1:00:13
this swelling in the heart tissue. And when there's swelling, the pressure from the inside is greater now.
1:00:19
And so now the blood, usually the pressure is greater from the blood coming in, but when that swelling happens, the blood can't get in anymore.
1:00:27
And that creates necrosis, tissue death. There's no oxygen, there's no nutrients to that area, and we get this
1:00:33
area of tissue death. And in "Baraldi's" work, that one scientist, he showed that he autopsied people with heart attacks.
1:00:39
And he showed probably, I think it was like half the cases over his career that the heart attack people had the tissue death people had was
1:00:47
in a totally different place than where the blockage was. So if it was because of this blockage restricting blood flow to an area,
1:00:54
they would correlate with that. - Now, mine was that way. I did have a blockage in the area where the tissue death was,
1:01:00
was downstream from the blockage. But in many people, according to "Baraldi's" work, that was not the case.
1:01:06
So these people likely had these “MINOCA” type heart attacks. So
1:01:11
it's interesting. This is way more convoluted than we think... And it also suggests an entirely different way of helping prevent
1:01:18
that type of heart attack, which has to do with balancing the autonomic nervous system and optimizing our metabolism and setting our circadian rhythm.
1:01:26
All this stuff that we could do. I want to come back to something you mentioned quickly earlier.
1:01:33
The fact that but being a type 1 diabetic makes you more susceptible to heart disease.
1:01:39
And when you went through and talked about your situation, having a heart attack,
1:01:44
you didn't bring that piece into being causative. So I'm curious if you think being type 1 diabetic
1:01:51
was part of your heart attack or not. -- It could have been in two ways.
1:01:56
Again, there's no way of really knowing, but theoretically, yes, it could have been in two ways. So my blood sugars,
1:02:03
no matter how hard I try, are never going to be as normal as someone without type 1 diabetes. Now, I know some type ones who achieve
1:02:10
definite levels of normalcy, but it's not the same for everybody. I know people that are doing the exact same thing as me,
1:02:16
and their blood sugars are completely normal. They were usually diagnosed later in life. - I was diagnosed at age 9.
1:02:22
And I think there are different types of type 1. So my blood sugar is never going to be as normal,
1:02:28
which means there is fluctuation which has been shown to cause artery inflammation. So that could have been I was
1:02:34
somewhat predisposed because of that. I already have this maybe low level artery inflammation,
1:02:40
especially with stress, which during stress it's very hard to control blood sugars.
1:02:45
But I eat a pretty low carb diet and that makes it very easy to control blood sugars
1:02:50
from my perspective. So that's one thing that could contribute. The other thing is that
1:02:56
being type 1 diabetic, especially when I wasn't controlling things back when I was younger,
1:03:01
the fluctuation in blood sugars can create more cortisol, or stress response. And over time
1:03:08
that can lead to this imbalance in the autonomic nervous system, which we just talked about,
1:03:14
mechanisms by which that can create issues. And there's some literature that shows that type 1 diabetics
1:03:21
are less able to handle the stress response of, say, an intense workout, which is exactly what I did before the heart attack.
1:03:29
So, yeah, there are different ways that it may have contributed or kind of set the stage for me.
1:03:35
And then the environmental factors pulled the trigger. - - But I don't know for sure.
1:03:40
And all I know is that I got to keep living the way that I'm living and protecting the
1:03:46
arteries the way I'm doing it. -- Let's come back to your paradigm and how you see
Structured water and heart disease, a fascinating connection
1:03:52
cardiovascular health and how it differs from conventional thinking.
1:03:57
And the part I want to zoom in on to start is this "EZ" water piece. So we have our endothelium, we have the glycocalyx,
1:04:06
and the way you see things, there's this other layer on top of that forming another boundary
1:04:12
with "EZ" water. So let's go there. Yeah, this is my favorite thing to talk about.
1:04:21
So - there are three things that I think there are three major changes over the last
1:04:26
hundred years or so when we saw this exponential rise in heart disease, coronary artery disease, plaque atherosclerosis,
1:04:34
that I think explain why it's happening because I said, it was rare in early 1900s
1:04:40
and it has to do with changing our environment... to an extent that has interfered with structured water formation.
1:04:48
So what is structured water? Many scientists have discovered that water has a fourth
1:04:54
phase that it can exist in. So we all know solid, liquid, gas, ice, water, steam,
1:04:59
but it actually has this fourth state and it's more like a gel. So you can think of like the consistency of raw egg white
1:05:07
or "Jello" or whatever. - It's kind of like that. And so again, different scientists have come across this over the years.
1:05:14
So it's got many different names. It's called structured water, exclusion zone water, fourth phase water,
1:05:20
bound water, different things. And so the most compelling evidence to me that most of the water in
1:05:27
our body is in this gel state is the fact that I feel like a gel. If I push my forearm or my thumb down or whatever,
1:05:34
the tissue gives and it bounces right back. Just like if I was pushing "Jello," it would give and bounce right back.
1:05:41
And so they have shown in the lab of Dr. "Gerald Pollack" and many other scientists have found
1:05:46
that there's this cell free layer next to the lining of the artery that no red blood cells or
1:05:51
anything really penetrate. That's right next to the lining of the artery. So if you just go into the "Google Scholar" and type in cell free layer,
1:05:59
you can read all the articles that come up that show this. And they didn't know what it was for a long time.
1:06:06
But "Gerald Pollack" and his lab at University of Washington has really delved into the structured water piece of it.
1:06:12
And so what happens is that when water holds energy, which water has this unique ability among liquids to hold energy,
1:06:19
and when it does, and it gets next to a water loving surface, a hydrophilic surface, which all biological surfaces are water loving,
1:06:26
then it will actually... change its structure a little bit. So what happens is oxygen or water is
1:06:32
an oxygen and two hydrogens and one oxygen and hydrogen come over here and one hydrogen is cleaved off
1:06:38
and those oxygens and hydrogens that are left team up with other oxygens and hydrogens and they form this lattice like structure,
1:06:46
they kind of this hexagonal structure that forms this planar like surface.
1:06:51
And this is why it's structured water, so to speak, because it has this structure to it,
1:06:57
but it's not rigid like ice... And so it line up next to hydrophilic surfaces and it forms
1:07:02
this stacked area. So there's this layer of gel. So now, this water... has some unique properties.
1:07:09
One, the reason that it's called exclusion zone water is because when it forms, almost nothing can penetrate it.
1:07:16
So it excludes anything that's not. It has an exclusion zone. The only things that can penetrate it are very small hydrated ions of minerals,
1:07:24
including precursors to nitric oxide, which come from the lining of the artery, and they can get through
1:07:30
the structured water and form nitric oxide. But... in Doctor "Pollack's" book, he notices that albumin,
1:07:37
which is the smallest protein in the blood, cannot penetrate it. And it's 1.8 nanometers in diameter.
1:07:43
So anything larger than that is not going to touch the lining of the artery if we have healthy structured water there.
1:07:51
And so lipoproteins are way too big, red blood cells are way too big. And the only thing that's small enough is
1:07:58
anything -- hydrated potassium ion, or smaller can penetrate the structured water.
1:08:03
So, remember back to "Virchow's” triad. He told us what causes clotting. - He told us that
1:08:09
damage to the lining of the artery, poor stagnant blood flow, and elements of blood being too stuck together are what cause clotting.
1:08:17
And that still holds true medicine today. That's still accepted as what causes clotting. And so
1:08:23
if we want to protect the lining of the artery from damage, we build structured water because nothing can penetrate it.
1:08:29
So that's the first thing. The second thing is that in "Dr. Pollack's" lab, they've actually shown that when you build structured water,
1:08:36
so structured water was a balanced molecule because it had two hydrogens and an oxygen,
1:08:43
but now one of the hydrogens is gone. And so the structured water becomes a very electronegatively charged
1:08:49
substance. - Because the oxygen is a bigger molecule. And so you have this negatively charged area, and those hydrogens that were cleaved off are concentrated
1:08:57
into the space next to it. And that's a very positively charged area in a positive next to a negative it's a battery,
1:09:03
which is why we have positive and negative ends on a battery. And they've actually done experiments in his lab where they form structured water,
1:09:09
and they plug an electrode into the positive and negative end, and it powers a light bulb. So you can create energy
1:09:14
from this. - And so they've also shown that you put a tube made of a hydrophilic solution or hydrophytic material into a water bath,
1:09:22
and you shine infrared light energy onto the water, and the water starts moving through the tube with no pump acting on it whatsoever.
1:09:29
So the energy created by structured water does the work of moving fluid. And they've actually proven that this happens in chick embryos.
1:09:37
They stopped the heart of a chick embryo, and they shine infrared light on the vascular system, and the blood continues to move with no pumping heart...
1:09:45
So this is really how the blood is moving. It's the main way the blood is moving. The heart does do a small amount of pumping,
1:09:51
but it is not responsible for the movement the blood primarily. And so that's "Virchow's" triad number two is have adequate blood flow.
1:09:59
And so infrared light building structure, water creates this adequate blood flow. The third part of "Virchow's triad is elements of blood being too sticky.
1:10:08
So they've also shown that structured water forms on the blood. Elements of blood, the red blood cells and lipoproteins
1:10:14
have what's called this zeta potential. And the zeta potential is just a negative charge that surrounds the red blood
1:10:20
cell or surrounds the lipoprotein... And things that have like charges repel each other. So if everything holds their negative charge of structured water around it,
1:10:29
they're never going to clump together. - And one of the biggest risk factors for heart disease is what's called
1:10:35
an "ESR" test, which is a measurement of how likely that your red blood cells are going to stick together...
1:10:41
And if you build structured water on them, they're very less likely to stick together. And so that keeps the blood very
1:10:47
thin, so to speak, less viscous. And we're keeping the blood thin not by interfering with clotting fact,
1:10:53
like a blood thinner does, but by building structured water on the arteries. And so... that's virtual
1:10:58
number three. That's how we prevent clotting and we build structured water in the arteries. So
1:11:04
what's the number one thing that builds structured water? There's lots of things that do, but the number one thing is based in Doctor "Pollack's" lab,
1:11:12
is infrared light exposure. And the reason this makes sense is because the sun, anytime it's up, is 40 to 50% infrared light.
1:11:20
So humans and all life is supposed to be outside. So what has happened over the last hundred years?
1:11:27
Three things. Those three things are in the 1950s, we invented fluorescent light bulbs.
1:11:33
So we went away from incandescent, which are high in infrared light. And we were also using fire before that and sunlight.
1:11:39
We were outside a lot. So we moved indoors under this bright fluorescent light, which is void of infrared light.
1:11:45
So we compromised our ability to make structured water. And that's the protection from the clotting that is atherosclerosis.
1:11:53
So that's step number one. You remove the protection or at least decrease the protection
1:11:59
- because of our lack of exposure. Second thing is, we added in all these toxin exposures and processed food diets.
1:12:05
That's what happened in the 50s. We started having this processed foods and all these different ingredients.
1:12:10
And even before that, seed oils were added in the early 1900s. So that didn't help. So then we added insult.
1:12:16
We removed the protection, added the insult. Now we're getting inflammation to the lining of the artery,
1:12:21
damage to it, and we're also not getting very good blood flow because infrared's not there, creating the blood flow in the structured water.
1:12:29
The third piece is, once we've caused the damage, we created an epidemic of diabetes
1:12:34
and insulin resistance and poor metabolic health, really, which can be present well before you're diabetic.
1:12:40
And insulin signaling and melatonin levels are critical for the healing of the artery once the damage has happened.
1:12:47
So if you interfere with those things, you take away the protection, you add the insole odds of damage,
1:12:53
and then you interfere with the body's ability to repair it. You get this epidemic of cardiovascular disease,
1:12:59
which is the number one... prevalence and number one cause of death in the world is cardiovascular disease.
1:13:05
And that's exactly what's happened. It's a combination of all those things. And so when you think about it like that
1:13:11
and you learn about what else is in the blood, looking at lipids and trying to micromanage a lipid panel.
1:13:17
Sounds ridiculous. And it is. And this is where it's gotten us. Heart disease is still the number one killer,
1:13:23
even though people are taking all these statin drugs and everything, because they're totally ignoring the actual causes of heart disease,
1:13:30
which is basically low energy to the body and lack of structured water formation. All right, so we want structured water in the body.
The heart is NOT a pump
1:13:38
Three main reasons. The triad protects the vessels, it keeps red blood cells from clumping,
1:13:45
and it promotes healthy blood flow through the body, correct? Mmhmm. Yep.
1:13:50
Talk more about the blood flow piece and the heart not being the primary mover of blood.
1:13:57
Again, reading your book, this is one of the things that was new information to me.
1:14:02
It's assumed in conventional medicine that the heart is contracting and pumping blood through...
1:14:08
the arterial system and the venous system. You've brought in this other piece,
1:14:15
but you also did say that the heart does contribute. How much would you say the heart is contributing versus this...
1:14:23
"EZ" water piece? Well, I don't know about exact ratios, but it's very little amount. So the heart does do some pumping,
1:14:30
but it's really no more than enough to move the blood through the chambers of the heart itself. - And the reason,
1:14:36
so we'll talk about the reason the heart is there too. But the heart is not
1:14:42
a pump in a sense that if people go back and listen to the first part of this whole thing, they'll probably realize that I never refer to the heart as a pump.
1:14:49
I always said the heart's contracting, not pumping, because that's what I firmly believe.
1:14:54
And there is a whole host of literature on this subject. There's a whole book called "The Heart and Circulation,"
1:15:01
written by Doctor "Branko Furst," that reads like a 200 page research... paper with tons of citations about how we have so much evidence
1:15:09
that the heart could not be, could not contract strong enough to pump the blood throughout the entire body.
1:15:15
--- And in school, -- we learned things like, --- the one way valves in the veins prevent it from coming back and
1:15:21
the movement of your muscles helps the blood move. But if you look at, there's studies that show...
1:15:27
if we look at the heart, it's a pressure propulsion pump, a pump that sucks in fluid from somewhere and forcefully
1:15:34
pumps it out somewhere else. The amount of energy that it uses, based off how much fluid moves through it or it moves,
1:15:41
quote, unquote, it's only about 15 to 30% efficient. Which, whether you believe we evolved or we were created,
1:15:49
that's not a very efficient way to create something whose job is to move fluid.
1:15:55
And so it can't be the main mover of the blood. So, and also just logically,
1:16:02
if I was going to pump blood from my toes to my head or to my chest, I wouldn't put the pump at the top of the hill,
1:16:10
I'd put it at the bottom and pump it up. So it doesn't make sense logically, but also
1:16:16
we have to ask ourselves -- how does water get from the roots of trees up to the leaves?
1:16:22
There's no heart in a tree so it has to move by different mechanisms. - And in Doctor "Pollack's" lab,
1:16:29
they've shown that in tree roots that destructured water formation happens and it moves water through the tree roots
1:16:36
and up against gravity, so to speak. So, yeah, there's no way that the heart could be the main mover of the blood.
1:16:43
And in fact, there's evidence that when the heart is being forced to be more of a mover of the blood,
1:16:49
it's because these other mechanisms are broken down in the body and that's when we can. That's one thing.
1:16:55
It's one small part of what can develop heart failure. The heart is being forced to be more of a pump than it's supposed to.
1:17:03
So if the heart is not the move we've shown and we've talked about the mechanism by which blood does move.
1:17:09
And so the heart actually operates more like a hydraulic ram, which I didn't know what that was.
1:17:14
When I first read that, I was like what's a hydraulic ram? So I had to go to "YouTube" and learn what a hydraulic ram was.
1:17:21
And the heart is actually, two hydraulic rams put together, the left side and the right side... And so a hydraulic ram
1:17:27
is a mechanism that is flow activated. So it has to have something flowing into it to be activated and to function.
1:17:33
And there's a few differences between the heart or one side of the heart and a hydraulic ram, but they're minor differences.
1:17:40
So in a hydraulic ram, water is flowing down into something from gravity, and then it fills up one chamber, and then when it fills it up,
1:17:47
it closes the valve, and that creates increased pressure, and then that pushes another valve open.
1:17:52
- And when it pushes that open, the water flows into the next chamber. And then when it flows up into that chamber,
1:17:59
flows up so much that it fills up, and the pressure created by that filling up closes the valve again.
1:18:04
That valve that opened to let it in, and when it creates that pressure, it flows out another valve and that
1:18:10
pressure that's created that, it pumps it out sort of. It's a hydraulic ram pump, but it's flow activated.
1:18:16
There's no force. And engineers can tell you how hydraulic rams work better than I can.
1:18:23
But I compare the analogous structures of a heart or one side of the heart and the hydraulic ram in the book,
1:18:29
and there are two differences. One is the heart doesn't have a spill valve in the first chamber.
1:18:34
And instead of... pressure building up and pushing back, there is a contraction of a ventricle
1:18:40
that it provides that little push. But again, it's just enough to push the fluid out of the ventricle itself.
1:18:47
And then once it gets back out into the arteries, the structured water mechanisms pick it up and it moves it along.
1:18:55
- There's even actual evidence that the blood has some magnetic properties. And so when a valve opens,
1:19:01
there's actually more of a suction happening out of the ventricles. It's not a, if it was a pumping thing, we would see the aorta, which is an arch.
1:19:10
If it was a pump like that, we'd see the aortic arch expand. Just like if I had a hose that was coiled up on the ground and
1:19:17
I forcefully turned on the hose, the hose would unravel. We don't see that.
1:19:24
When you look at angiograms and visualization of the aortic arch,
1:19:29
when the heart is supposedly forcefully contracting, it actually curls up like this, like there's a suction happening.
1:19:36
And that's what happens is that because of that... magnetic attraction -- when the ventricle valve opens,
1:19:42
it sucks the blood out of it and creating this contraction effect -- of the aortic arch.
1:19:48
And so that's how the heart works. It's this hydraulic ram pump, so to speak. But it has to be flow activated.
1:19:55
So if the flow mechanisms are broken down, like if we're not getting enough structured water and we're not
1:20:00
grounded and we're doing things that interfere with blood flow, we have a lot of bound up fascia in our body,
1:20:06
those types of things, then... the blood is moving more stagnantly through the heart and the heart's being forced to be more of a pump than it's supposed to be.
1:20:14
And that puts a lot of strain on the heart. And it's one thing that contribute to the development of heart failure
1:20:20
where instead of the heart contracting in a spiral like nature, it starts to balloon out and become this basketball shaped thing,
1:20:27
this cardiomyopic situation. So the heart is not a pump. There's plenty of evidence that suggests that.
1:20:35
And so what is it then? What's its purpose? And in my opinion, there's three different roles of the heart and the body.
1:20:41
None of them are to forcefully move blood. One is another way that we energize
1:20:47
water is by vortexing it, which means swirling it around or swishing it around in some way.
1:20:52
And this makes sense because in nature water is always moving, it's always being switched around,
1:20:58
it's crashing on the beach, it's being evaporated and precipitated, it's flowing in rivers.
1:21:03
Nature is not keep water stagnant and if it does get stagnant, like in a pond or a puddle, it gets dirty.
1:21:10
And it's not really, it builds a bacteria. So water's always supposed to be moving. However,
1:21:16
when you move it through pipes in our blood vessels, it doesn't get switched around very much.
1:21:21
So you have to insert a mechanism into the middle of those pipes, which is a peculiar place for a pressure propulsion pump to be.
1:21:28
Would be in the middle because it's not a pressure propulsion pump and it's there to...
1:21:34
mix up the blood. Pretty much it's a vortexing hydraulic ram. And that's exactly what we see.
1:21:39
When the blood is flowing into the heart, it flows in like a vortex, kind of flows in like that,
1:21:46
Kind of like when you drop one of those quarters in those things and it goes around like that,
1:21:51
it flows in like that... The heart is actually one big band of muscle that's wrapped up on itself.
1:21:57
So when you unfold it, it's this one big continuous band of muscle. You wrap it up on itself and when it contracts,
1:22:04
it spirals like this. And so it's vortexing the blood. Also, when the blood flows out of the valves, it's eddying on both sides.
1:22:13
So it's just creating this disruption of flow to vortex the blood and energize it in the presence of oxygen.
1:22:18
So there's no mistaking or there's no coincidence that the blood comes into the heart, gets vortex, goes to the lungs,
1:22:25
comes back to the heart, gets vortexed again to get more in the presence of oxygen.
1:22:30
And then so that when it gets out to the bloodstream, it is more energized so that
1:22:36
it can form the structure of water and create blood flow. So in a way, indirectly, the heart is responsible somewhat for the flow of blood,
1:22:43
but it can't do it itself. It needs the right environmental stimuli to do it as well. So we need infrared light and grounding and things.
1:22:51
And so that's the first rule of the heart. Second rule of the heart is that that
1:22:56
in times when we exert ourselves, the tissue demand for oxygen and nutrients is so high that if the
1:23:01
heart wasn't there to actually slow the flow of blood, all the blood would rush over to the arterial side and the venous side
1:23:07
wouldn't have enough pressure and it would collapse. And so the heart is actually... slowing the flow of blood and exertion.
1:23:14
And there's some very eloquently done studies showing this in endurance athletes, that indeed, whenever they're exercising,
1:23:20
the heart is kind of a damming up organ. It's stopping the flow of blood. So people think endurance athletes have... this
1:23:27
hypertrophic heart. The heart can get bigger in size and muscle, and it's actually because they've shown that it's better
1:23:34
at stopping the flow of blood than forcefully pumping it. So it's kind of like a bigger catcher's mitt
1:23:41
to catch the flow of blood and then vortex it. It's there to stop the flow of blood during exertion.
1:23:48
And then the third aspect of this, or the third role of the heart, is that
1:23:54
the heart has the highest electromagnetic field of any organ in the body. It's letting off this electromagnetic field.
1:24:00
And so all organs let off this field, and organs with the highest amount of mitochondria let off the biggest field.
1:24:06
So the eyes, the brain, and the heart have similarly high amounts of mitochondria.
1:24:12
- So why is the heart giving off the biggest electromagnetic field? It's because there's evidence that when you vortex, when you spiral
1:24:20
something in an electromagnetic field that's already exist, it amplifies the field exponentially.
1:24:26
And so, because the heart is the moving organ that's spiraling in our body like this over and over again,
1:24:31
it's amplifying that field so you can. Researchers have detected this electromagnetic field from,
1:24:37
I think, up to 16ft away. And so it's the organ... that we reach out into our environment...
1:24:43
with, which is why we attribute all of our emotions and our feelings and everything with our heart.
1:24:50
We feel things with our heart because it's what's allowing us to connect to other people's field
1:24:56
and also connect with the environment around us. It's measuring coherence in the body,
1:25:01
so it's measuring how synced up and communicating our body is. And all of our cells are signaling,
1:25:07
and it's a sensory organ. It's sensing our emotional state and relaying that information to the brain,
1:25:14
which is why we say, I love you with all my heart, and I love you with all my brain.
1:25:19
So it's an incredibly fascinating organ that I don't think we fully understand.
1:25:26
And it's incredibly important to understand if we ever want to tackle heart disease.
1:25:31
Coming back to the first purpose of the heart, the vortexing... gets me thinking about structuring water outside the body before we drink it.
1:25:39
And there's a number of different devices. There's devices that spin water and create a vortex.
1:25:46
There's wands you can use in your water... There's all kinds of different devices.
1:25:51
So given the importance of structured water, and you've talked about getting sun and grounding,
1:25:57
ways we can enhance the water already in our body. What about water we're going to ingest
1:26:03
and using some of these modalities beforehand? Any value there? Definitely. So in Doctor "Pollack's" lab,
1:26:11
he's shown that... the toxin, the herbicide glyphosate, directly interferes with structured water formation.
1:26:19
So having clean water and I imagine that's not the only toxin that would do that. It's just the one that he's tested, acid.
1:26:26
So having toxin free water is very, very important. So getting rid of the toxins in your water,
1:26:32
adding minerals to it, because the hydrated ions of minerals, they're hydrated, they're kind of the structured water forms around those minerals.
1:26:39
So adding minerals back to the water and then yes, you can treat it in some way, so to speak, or energize it, I would say,
1:26:45
because I would argue that you can't really drink structured water because it would technically be in a gel state if it was structured water.
1:26:51
So you can't really drink it. Be like trying to drink "Jello." But you can energize the water.
1:26:56
So then when it gets next to a biological surface it, yes, more readily structures itself. And
1:27:02
so, yes, you can vortex it with these different devices that will do that. --- The most low tech way to do it is get
1:27:09
a little wine glass and just swirl it around if that's all you can do, it's better than nothing.
1:27:15
- You could put it in the sun. - put it in contact with the air. I know some people have these little
1:27:20
"PEMF" pads that mimic the electromagnetic field of the earth or something similar, and they put it on that...
1:27:26
There's ways that you can create energy or coherence to the water so that it's more likely to structure itself when it gets in use.
1:27:32
So I think it is worth doing that. But also remember that if you don't have devices that will do that,
1:27:38
there's low tech ways to do it. You can put it in the sun, or you can drink clean, mineral rich water and then put your body in the environment
1:27:45
that helps structure the water. Because I think that hydration - for the vascular system
1:27:50
drinking water for hydration, is important for the vascular system. But full body hydration I think is more about metabolic health,
1:27:57
because... mitochondrial function or optimal mitochondrial function, you're creating metabolic water in the cells.
1:28:02
So if you have poor mitochondrial function, you're dehydrating your cells. - But I think for the vascular system,
1:28:08
drinking water is important for maintaining hydration in the vascular and lymphatic systems and things like that. --
The best ways to structure water in your body
1:28:15
You mentioned "PEMF" mats using one of those or one of these mats that you can plug into your outlet...
1:28:22
and it connects to the grounding part of the outlet. How do those compare from your research to going outside and actually
1:28:31
putting our feet on the grass? Yeah, nothing's going to be as good as going out onto the earth
1:28:37
and putting your feet directly on the earth as far as conducting electrons from the earth.
1:28:42
- And a "PEMF" manages different frequencies that are trying to mimic or be more
1:28:47
compatible with the electromagnetic field of the earth, the "Schumann" resonance. So both of those things,
1:28:53
you're in contact with both of those things. --- We're in contact with it right now. Even though my feet aren't on the earth, I'm in contact, I'm in that field,
1:29:02
but I'm closer to that field if I'm on the earth and I'm in direct contact with it conducting electrons.
1:29:08
If I'm in contact with it. My feet are on a grounding mat right now. Because when I am indoors... and I can
1:29:15
be grounded, I try to be. So I have grounding sheets, grounding mats I don't know that anybody's ever looked at compared to -- how much I'm conducting
1:29:23
with feet on the earth based versus a grounding mat. - And I'm sure it would be different for different homes.
1:29:30
But yes, they definitely work. Lots of the studies on grounding were done with grounding mats inside and they
1:29:36
saw physiologic effects of reduction in inflammation and blood flow and increase data potential on red blood cells
1:29:43
with those mats. But I always default to the original way it was done. I think that's always going to be best.
1:29:50
And for somebody that wants to go stand in the grass or use one of these mats if they're on their computer,
1:29:56
anything we need to be aware of when it comes to say, underground wiring or being close to certain structures that are emitting
1:30:06
electricity into the ground. Basically, what do you look for
1:30:13
when you're grounding to make sure that ground is safe and
1:30:18
health promoted? Yeah, so I wouldn't, -- if you knew I wouldn't necessarily ground
1:30:25
on an area where there's big power lines underneath you, I don't know that that would be necessarily
1:30:32
less harmful or more harmful. -- But --- it doesn't seem like it would be,
1:30:38
like it's interfering with some conduction. But I have heard some theories that being grounded actually protects you from
1:30:47
electromagnetic fields in some sense. So yeah, definitely being -- submerged in natural water,
1:30:54
that's very protective against. You're very grounded in that state and that's protecting you from "EMF."
1:31:01
But yeah, but then there is things like dirty electricity. So dirty electricity is basically
1:31:07
this crazy "EMF" field and all these different electricity is coming from all these different places can kind of find its way into the wires in your home.
1:31:15
And since there's "AC" current in the home, it's kind of this pulsating... thing. And so if you're worried about that, you can buy dirty electricity filters
1:31:23
that you can plug into the wall that kind of eliminate that dirty electricity, cut off the circuit,
1:31:28
so that when you plug in the grounding prong, you're not worried about getting any of that through that wire,
1:31:33
because the grounding prong is not wired to any electricity. It's just going to the ground.
1:31:39
But it can, it's a wire that's conductive, so it can absorb some dirt electricity if it's around. So if you want to avoid that, you can get those filters.
1:31:46
But like - my home, I've had it tested by a building biologist, and they said their electricity wasn't that big of a deal.
1:31:53
So I don't worry about it too much. Right now with the grounding mat that I'm using, but when I sleep at night
1:31:58
I turn it off electricity to the bedroom altogether, and then the grounding sheets are plugged into the wall.
1:32:03
So I'm not worried about it at all at that point. But yeah, there are concerns sometimes, especially
1:32:11
older homes, I'd say that may have some poor wiring or areas
1:32:16
in cities where there's lots of "EMF" and different electricity everywhere those can be concerns.
1:32:23
But yeah. All right, so going over the areas where you're grounding bed sheet
1:32:28
at your computer right now, your feet are grounded. Do you have any other grounding mats throughout the house?
1:32:35
- No, I just have one, and it kind of follows me around the house.
1:32:40
And then I got side. I was offensive game. But that's the other thing too, is -- on grass or dirt,
1:32:46
you're very grounded. But also if you have a concrete patio that's maybe 4 or 5 inches or less and it's directly on the earth,
1:32:53
you're conducting through that somewhat. I have a little grounding tester that I can test if -- my circuit is complete
1:33:00
with the earth, but also... the leaves of a tree. You can touch the leaves of a tree and you're grounded.
1:33:07
Patio stones, those types of things. The beach, the sand. Places that are not grounded are asphalt...
1:33:13
mulch. It's dead wood. Not very grounded, not conductive... places like that. So, yeah,
1:33:19
but as much time as you can spend feet on the earth, it's a good thing.
1:33:24
All right, so number one is getting your feet right on the earth. If somebody wants to be more prescriptive with this.
1:33:32
How much grounding is ideal?.. It sounds like you're doing quite a bit with your hacks and doors. Yeah.
1:33:40
But now that we're moving into the practical. How much would you say is a good amount to aim for per day?
1:33:48
It's hard to say. I don't know that anybody's really measured. Well, I do know of self experiments where people will look at
1:33:55
their live blood analysis and they'll look at it beforehand and their red blood cells are clumped together.
1:34:01
They see this rouleaux formation on there and then they'll go out and do 30 minutes of grounding and the red blood cells
1:34:07
are perfectly evenly spaced after that. And then about, I'd say an hour after grounding, when they've stopped grounding,
1:34:14
they're back to being fairly --- not as bad as they were to begin with. But they're pretty clumped together.
1:34:21
Now, dthat's just an N of one experiment. And I also don't know much about that person's -- environment they went
1:34:27
back into after they grounded. I don't know if there's a high "EMF" environment or whatever it was.
1:34:33
- But that gives people a sense of how long that's going to last. You got about an hour.
1:34:39
If you spend 30 minutes grounding to where it's going to go back to a more...
1:34:44
coagulative state, so to speak. Which is why the indoor grounding tools are useful because I can be
1:34:50
grounded as much as possible. Even if it's less of a conduction than outside, it's still something.
1:34:55
- And grounding sheets are very useful because I can be grounded all night long. -- So yeah, and then minimizing
1:35:02
your exposure to things that have been shown to steal the charge, break down structural water. So wireless routers have been shown to do that.
1:35:09
- Artificial light is very confusing. We want the infrared light that's very building the structured water.
1:35:16
So I have incandescent bulbs in the house. If I -- am using light bulbs, I try and be outside
1:35:22
red light panels, infrared sauna. I try and boost my infrared exposure so I don't have to live outside
1:35:28
necessarily. But I spend my time outside as much as I can. - But yeah, it's doing those types of things.
1:35:35
When it comes to infrared sauna, I know there's near infrared and far infrared. I personally have a far infrared at home.
1:35:43
From reading your book, I know that sweet spot for creating structured water is 3,000 nanometers.
1:35:49
Is that part of the far infrared spectrum? Yeah, it's far infrared. Yes. That's the farthest
1:35:54
wavelength of infrared that would come naturally. So that's what they tested in Doctor "Pollack's" lab,
1:36:00
that farthest wavelength. And so it's basically the spectrum of electromagnetic light.
1:36:06
So you have infrared through "UV" that's the only light that we're ever supposed to get from the sun.
1:36:11
All this other artificial light, whether it's wireless signals or the blue light, whatever,
1:36:16
it's either processed light or it's outside that spectrum. And so "UV" also makes structure water. But it was the least efficient.
1:36:23
And then the far other side of the spectrum, the far infrared, was the most efficient at building the structured water.
1:36:30
So that's why I have a far infrared sauna. And the sauna I use. - - it's been tested. And it's
1:36:36
as far as I know, which I don't know everything about every sauna, but as far as I know, it
1:36:41
emits the most far infrared light of any far infrared sauna out there. It's the one I use to reverse the plaque in my leg
1:36:49
or the one I use while reversing the plaque in my leg. So, yeah, technically I would think far infrared is the best for building structured water.
1:36:57
But near infrared will do it. Red light will do it. All the wavelengths of light will do it, aside from process blue.
1:37:04
There's this discussion to have. People will tell me all the time, well, if processed light is bad, then a far infrared sauna is processed light.
1:37:11
It's one wavelength. It's always supposed to be balanced with all the colors and wavelengths. And I was
1:37:16
you're exactly right. And if I was to overuse that process light, then I think it would be harmful if we had chosen
1:37:23
red light or infrared light, which it's not visible. So we couldn't light our homes with infrared light.
1:37:28
But if we had chosen red light to light our homes, I think we'd all be talking about the harms of red light at this point.
1:37:35
But it -- didn't make sense because red light's not that bright and didn't really make sense to do that.
1:37:40
So we're talking about the harms of processed blue light and light that is very high in blue because
1:37:46
you can look at, there'd be a threshold as you do the red light laser therapies for treatment of scar tissue and things like that in your body.
1:37:53
- There it's beneficial, beneficial, beneficial gets a certain point and then it's negative.
1:37:58
So if we overdid this, I think it would be harmful. But the reason it's seen as a therapy, these red light panels, near infrared,
1:38:04
far infrared, is because we're so deficient in infrared light because technically we're supposed to be outside...
1:38:10
during the day, exposed to it all day long. Even if we're in the shade, we're getting infrared reflecting off the grass and stuff.
1:38:17
And so we're just so deficient in it that it seems like a therapy, and I think an important therapy to use
1:38:23
if you can't be out in the sun all day long. But I think that... if you -- look at all the statistics and the trends in diseases,
1:38:31
every disease gets worse in the winter. We have worse heart attacks, worse deaths from cancer.
1:38:38
Every disease gets worse. And when you think about the fact that it's harder to get outside.
1:38:45
- People just get less sunlight in the winter, depending on where you live, then it makes sense.
1:38:51
We're getting less structured water, which plays a role in every chronic disease.
1:38:56
And that's causing an issue when in the winter, among other things, we also eat a bunch of processed foods around the holidays and all
1:39:04
this other stuff that we do but that's one contributor. That's the light story.
1:39:09
Okay, so far to build structured water, infrared light, grounding,
1:39:15
what other areas do you try and hit on within a day? - Infrared light and grounding are the main ones...
1:39:22
we have removed ourselves from in our modern way of life. So those are the main ones to add back.
1:39:27
But then like I mentioned, there are things that tear down structured water. So we have the wireless signals
1:39:33
in Doctor "Pollack's" lab a wireless router, just a normal wireless router, interfere with structured water formation 15 to 20% reduction.
1:39:41
- So we're inundated with these signals these days. And you're never gonna get rid of them all,
1:39:47
but it's important to mitigate them when you can stay as far away from. So the closer it is to you, the more harmful it is.
1:39:53
So keep it as far away from you as possible. Put the wireless router somewhere else in the house. Don't put your cell phone next to you.
1:40:00
Don't use a bunch of wireless headphones and all that kind of stuff. If you can avoid it and then eliminate it when you can.
1:40:06
When you sleep, you don't need it. Turn it off. I sleep in an "EMF" blocking canopy. That gets rid of all of it,
1:40:12
all the cell towers and everything from around me... So yeah, you can go as far down that rabbit hole as you want.
1:40:18
So that and then I mentioned toxin exposure before. Those things, toxins have been shown.
1:40:24
-- It makes sense. So anything that we think of that causes oxidative stress something that's trying to steal an electron from somewhere because it
1:40:32
wants to be paired with electrons, these free radicals, anything that can do that. Well, guess what a very
1:40:38
-- robust amount of electrons is stored in structured water. So if you have this oxidative stress, a lot of these molecules
1:40:45
toxins or reactive oxygen species from damaged mitochondria, things like that, poor metabolic health,
1:40:51
then you have a lot of these things that can potentially steal energy from structured water and break it down.
1:40:59
So that's an issue. So anything like that, and like I said, it hasn't been directly tested,
1:41:05
but I wouldn't be surprised if heavy metals and plastics and herbicides and pesticides and all these different things...
1:41:11
all steal the body's charge of structured water and break it down. So that's the other thing is removing the things as much as you can
1:41:19
that tear down structured water. And the good news within this is a lot of the things you're talking
The REAL energy currency of the body (Not ATP)
1:41:26
about to build structured water and to protect against breaking our structured water
1:41:32
are things that are part of what people in our world know as a healthy lifestyle.
1:41:38
It's just looking at it from this other angle of why those things might be helpful
1:41:44
because they're acting on the water in our body. Yeah. So if you, if you take a big picture
1:41:50
this is ultimately what I've come to discover or think is that life is just
1:41:56
it's all about harvesting energy from the environment and using it to create order. So
1:42:02
the universe abides by the laws of thermodynamics, which is energy is never created or destroyed. It's only passed between two things.
1:42:08
And that things with concentrated energy tend toward disorder, entropy. And
1:42:14
what life is harvesting energy from the environment and temporarily defying the law, the second law of thermodynamics,
1:42:20
and maintaining order in a system, in an organized system, while that thing is alive.
1:42:25
And then once we die, it dissipates again. The energy dissipates. But while we're alive, we want to maintain that order.
1:42:32
And if you don't have enough energy in the system to maintain the order, you get disease, things start to break down.
1:42:39
And so the first place that seems to lose its order or the body seems to sacrifice is the lining of the arteries.
1:42:45
Which is why when we put ourselves in a situation that has deprived of our energy sources, infrared light, grounding,
1:42:51
and then we've added these things to steal those energy sources. - Then the vascular system is what suffers the most. And that's why we have an
1:42:58
epidemic of cardiovascular disease --- in this country, especially in America.
1:43:03
So we have to put our body back into a situation where it can harvest energy from the environment. People think that we get all of our energy from food,
1:43:10
and that's absolutely false. There are scientists that estimate we should only get about a third of our energy from food
1:43:16
and the rest should come from our environment. And we have the ability to conduct electrons, which is what we're harvesting in the chemical bonds of food.
1:43:23
We're trying to take the electrons from them and transit point electron transport chain and make "ATP"
1:43:28
which is not the energy currency of the cell, but it's very important. Anyways, that's a different conversation though.
1:43:34
And so we're trying to get electrons from the environment, and you can conduct the electrons directly from the earth,
1:43:41
but we also have "DHA" and melanin in our skin that takes sunlight and convert it directly to electrons.
1:43:48
And these processes are there for a reason. And so if you live inside all day, out of the sunlight,
1:43:53
you're depriving your body of those electrons. So if you don't give your body energy, don't expect it to be able to maintain order.
1:44:01
That's basically the, the big picture fundamental understanding of it. All right, we got to come back to the "ATP" piece.
1:44:09
He mentioned quickly there the fact that it's not the energy currency of the body,
1:44:14
which science has, - it's a common understanding within the scientific world that it is.
1:44:22
Talk about how you see that. This is something I discuss in detail in my most recent book I came out with back
1:44:27
in August. It's called "Pain Sense" where I talk about the chronic pain epidemic. - But yeah, so there's
1:44:34
this, we're all taught in 8th grade biology that the goal of the mitochondria is to make "ATP," and "ATP"
1:44:40
is the energy currency of the body. However, there are issues with that in that this one scientist,
1:44:47
many scientists actually, but mainly this scientist named "Gilbert Ling."
1:44:53
experimentally and mathematically proved that I don't care how many mitochondria you have in a cell,
1:44:59
there's no way that it could make enough "ATP" to power just the sodium potassium pump,
1:45:05
if the sodium potassium pump even exists. And he was there's just no way to make that much.
1:45:11
And especially during cell division, that's said to take up 40% of the cell's energy to do that.
1:45:17
How's it supposed to do that if it can't even make enough to power the sodium testing pump?
1:45:22
And it's because... energy is, is stored in "ATP." There is an energy bond in the, in the chemical,
1:45:28
chemical energy bond that is storing energy in "ATP." But all it's really used for...
1:45:34
is to unfold proteins in the cell. Think of -- what does structured water need?
1:45:40
All of our cells are made of structured water. That's why we feel like a gel. If I poke myself.
1:45:47
And what structured water needs is energy, an energy source. It needs -- surface area.
1:45:52
It needs a spot to a hydrophilic surface to deposit itself on and become structured water.
1:45:58
And so if you roll up a ball of tinfoil, real tight that's very small surface area.
1:46:03
But if you unfold that tin foil, you create a lot of surface area. And so "ATP's" job.
1:46:09
The reason we pass electrons down electron transport train and then pass hydrogens the other way and go to complex 5 and make "ATP"
1:46:15
is so that those "ATP" can unfold proteins. Because when they unfold proteins, water structures on it and that's where the energy for our bodies
1:46:23
is held in structured water. It's very electronegatively charged. So when you think about building structured water, you're charging your body like a battery.
1:46:31
Remember I told you structured water creates a battery. It creates this thing that does work in the body.
1:46:37
And so that's where it's being stored. So "ATP" is still critical because we need surface area to build structured water,
1:46:44
but it's not the energy currency of the cell. And then further from that there's "Gilbert Lang" also has his
1:46:50
association induction hypothesis, which basically explains that because they've done experiments where you remove
1:46:56
the cell membrane of a cell and the solute distribution. Anybody who's familiar with cellular physiology knows that the potassium is
1:47:04
concentrated inside the cell and sodium is concentrated outside the cell. And we talked about how exclusion zone water...
1:47:10
excludes anything that's sodium or bigger and includes things that are potassium and smaller can get in
1:47:16
and that's exactly what we see. Potassium and chloride are inside the cell and sodium is excluded outside the cell.
1:47:23
And they did experiments where they removed the cell membrane. It stayed exactly that way because the structured water is still there
1:47:30
and the cell membrane is just... there kind of, I guess, holding it in a somewhat structure.
1:47:36
- And so that's how we get this solute distribution. And then "Gerald Pollack" wrote this fascinating book called "Cells,
1:47:43
Gels and the Engines of Life." That explains how what we think of as an action potential that does physiology,
1:47:50
that triggers for physiology in the body or the trigger of a cell to divide or do cellular physiology
1:47:56
is all driven by what he calls phase transitions, which is basically think of phases of water.
1:48:02
It's the structured water going back to a liquid state. And that creates a decreased voltage for a second because the voltage
1:48:08
is held by the structured water. You lose the structured water, you lose the charge and then it comes back.
1:48:14
But that change in voltage triggers for cell division and for protein making and all this different type,
1:48:19
it provides the energy to do that, the signals for that. He talks about how phase transitions are responsible for muscle contraction
1:48:26
and all these different things. So it's entirely different framework of looking at physiology. But it makes sense when you look at the big picture of how
1:48:34
life is on the planet. We're all trying to gain energy and we're trying to store that energy in structured water...
1:48:41
And we need other things to do that, but that's what provides the energy for life. And so why are we sick?
1:48:47
We've removed ourselves from those energy sources, added things that interfere with or that insult the energy source.
1:48:53
And then once the damage has happened, we've interfered with the body's ability to heal from it through
1:48:59
disrupted circadian rhythm and poor metabolic health. That's there it is right there.
1:49:04
Can we measure structured water at all? I don't know about in the body, but I know that in Doctor "Pollack's" lab,
1:49:11
they've, they've measured the amount that will form on a hydrophilic surface and
1:49:17
they've taken different things to make it build more and measured how much it will build it. Infrared,
1:49:23
far infrared light was the best at doing it. But yeah, I don't know.
1:49:28
I guess by proxy you could look at markers of inflammation. Inflammation is just low body charge in my opinion.
1:49:35
Inflammation just means too much, we've lost our alkalinity There's tons of studies that show that a change in "pH" of an area
1:49:44
is an indication of inflammation. So when the cell loses its slightly alkaline "pH,"
1:49:50
which means alkaline means a little bit more negative, which means less structured water.
1:49:56
You've lost your alkalinities. That's what inflammation is. So markers of inflammation could be
1:50:02
an indication that you're losing or you have low structured water in your body... -- You could say
1:50:07
an erythrocyte sedimentation rate, where you look at that and the red blood cells are clumped together.
1:50:13
There's low charge there. There's not enough charge to keep the red blood cells evenly spaced.
1:50:19
You could consider that by proxy a measure of structured water. But yeah, there's no
1:50:24
device that will measure the structured water on you. But actually that's how "MRIs" work.
1:50:30
They align the water molecules in a way that they can take an image through it. And
1:50:35
"Gilbert Ling's" work with structured water is what led to the development of "MRI" machine. And he was like, are you not interested in
1:50:41
the other things that this shows us? And they're no, no, we just want to make an "MRI" machine.
1:50:47
That's the story he tells in one of his books. So, yeah. Doctor "Hussey," before we wrap up, I want to bring diet into this.
1:50:56
I know you're a fan of the low carb diet, ketogenic diet, but let's tie that into the structured water piece...
1:51:03
and how they interact. Well, I like low carb diets and technically, I guess ketogenic diets, I think they're fine,
1:51:11
especially for someone like me who's type 1 diabetic trying to control blood sugars.
1:51:17
But I'm not myopic about diet. I think that we should eat whole foods. That's the most important thing.
1:51:23
And we should eat in a way that creates metabolic health... What I mean by metabolic health is that
1:51:29
your body can easily go back and forth from burning glucose and fatty acids
1:51:34
reasonably fast. - So if you eat a high carbohydrate diet for a long time, you lose the ability to make ketones
1:51:40
and you have to kind of restrict carbohydrates and force your body to up regulate those mechanisms to do it again.
1:51:46
So that's not a state we want to be in... So we want to create metabolic health. And the easiest way to do that is eat whole foods.
1:51:52
I think animal foods are critical to our health. One of the reasons being is that we just talked about how "DHA" which is an omega
1:52:00
3 fatty acid only found in animal foods, is critically important for you taking sunlight and making electrons from it.
1:52:07
So specifically in seafood we get high amounts of "DHA" and grass fed animal products and things like that.
1:52:12
So I think that's very, very important. And the reason
1:52:17
that's related to structured water is because we all learn again about "ATP" in cellular physiology. We learn this, the most important thing.
1:52:24
That's the reason the mitochondria there make "ATP." But in reality the mitochondria make "ATP." They make reactive oxygen species which are
1:52:32
important signaling molecules that tell you about metabolism and lots of different other things in the body.
1:52:38
There's some fascinating quantum aspects of reactive oxygen species. - But also you're making infrared heat.
1:52:45
We all know that we're emitting body heat. So there's your structured water former. Right there is if you have a healthy metabolism,
1:52:52
you're making structural water by releasing the heat. And you also make water as a byproduct.
1:52:57
I remember being taught. Oh, those are just byproducts of metabolism. - The "ATP" is what's important
1:53:03
when in reality the water is probably the most important. And so that's perfectly
1:53:09
energized, deuterium depleted water that will go right into the cell and stick to a protein,
1:53:14
become structured water on a protein. So that's when I say hydration and the cellular level is more about
1:53:21
metabolism that's what I mean. You're going to hydrate yourself when you take those electrons
1:53:27
down electron transport train, you get water as a product of it.
1:53:32
So, yeah, that's why that's super important to have that metabolic health and eat a
1:53:37
diet that creates metabolic health. But I'm not dogmatic about what that should be.
1:53:42
I'm a huge fan of sending your diet around... animal proteins that's going to create satiation and give you,
1:53:49
because it's giving you protein, vitamins and minerals... and give you a good source of
1:53:55
efficient energy, which is fatty acids. And then create variety with plants if you want to...
1:54:00
But I would advise to eat seasonally because I do think that there are signals given to us from food,
1:54:06
from our local and seasonal environments that tell our physiology different things to do. -
1:54:11
- It's also just better for a lot of different reasons. I think the food's more nutritious.
1:54:17
There's generally less toxins and that type of stuff. So, yeah, I eat whole foods.
1:54:23
Centered a diet around animal protein. Create variety with plants in season. All right, we're going to leave it there for today.
1:54:31
Really enjoyed the conversation. You've broken a lot of paradigms, gotten us thinking out of the box
1:54:38
and yeah, it'll be interesting to see how all this plays out. Yeah, thanks for having me on. We're going to link up your books, your social media, your website,
1:54:45
everything in the show notes. Thank you. Thank you. Now that you're done, you're going to want to stick around here and catch this other incredible episode.
1:54:53
You don't want to miss it. I'll see you over there. And I started looking into it and a bit like the film "The Matrix" where you take
1:55:00
the red pill instead of the blue pill, and you suddenly realize, at least in this area of medicine,
1:55:06
it's all just started to fall to pieces